Physiological Consequences of Compartmentalized Acyl-CoA Metabolism

J Biol Chem. 2015 Aug 14;290(33):20023-31. doi: 10.1074/jbc.R115.663260. Epub 2015 Jun 29.

Abstract

Meeting the complex physiological demands of mammalian life requires strict control of the metabolism of long-chain fatty acyl-CoAs because of the multiplicity of their cellular functions. Acyl-CoAs are substrates for energy production; stored within lipid droplets as triacylglycerol, cholesterol esters, and retinol esters; esterified to form membrane phospholipids; or used to activate transcriptional and signaling pathways. Indirect evidence suggests that acyl-CoAs do not wander freely within cells, but instead, are channeled into specific pathways. In this review, we will discuss the evidence for acyl-CoA compartmentalization, highlight the key modes of acyl-CoA regulation, and diagram potential mechanisms for controlling acyl-CoA partitioning.

Keywords: acyl-CoA; acyltransferase; fatty acid metabolism; fatty acid oxidation; phospholipid; thermogenesis; thioesterase; triacylglycerol.

Publication types

  • Research Support, N.I.H., Extramural
  • Review

MeSH terms

  • Acyl Coenzyme A / metabolism*
  • Acylation
  • Animals
  • Cell Compartmentation*
  • Culture Media
  • Homeostasis
  • Mice
  • Mice, Knockout
  • Signal Transduction
  • Substrate Specificity
  • Thermogenesis

Substances

  • Acyl Coenzyme A
  • Culture Media