CD114 Expression Mediates Melanoma Tumor Cell Growth and Treatment Resistance

Anticancer Res. 2015 Jul;35(7):3787-92.

Abstract

Background/aim: Melanoma tumor cell sub-populations expressing a variety of specific molecular markers have been identified. We hypothesized that expression of CD114, the cell surface receptor for granulocyte-colony stimulating factor (G-CSF), would be associated with melanoma tumor cell growth and response to treatment.

Materials and methods: We determined the expression of CD114 expression in tumor cell lines by flow cytometry. We separated melanoma tumor cells into CD114-positive and - negative populations by fluorescence-activated cell sorting (FACS) and measured cell growth and responses to temozolomide and etoposide and the anticancer agent nifurtimox.

Results: All tested cell lines demonstrated a sub-population of cells with CD114 surface expression. CD114-positive sub-populations grew faster than CD114-negative ones and demonstrated resistance to temozolomide, etoposide, and nifurtimox.

Conclusion: CD114 expression defines a sub-population of melanoma tumor cells with altered growth and resistance to treatment. Further studies on the role of CD114 in melanoma pathogenesis are warranted.

Keywords: CD114; G-CSF; melanoma; nifurtimox.

MeSH terms

  • Antigens, CD / genetics*
  • Antineoplastic Agents / pharmacology
  • Cell Line, Tumor
  • Cell Proliferation / drug effects
  • Cell Proliferation / genetics
  • Drug Resistance, Neoplasm / genetics*
  • Flow Cytometry / methods
  • Granulocyte-Macrophage Colony-Stimulating Factor / genetics
  • Humans
  • Melanoma / drug therapy
  • Melanoma / genetics*
  • Melanoma / pathology*

Substances

  • Antigens, CD
  • Antineoplastic Agents
  • Granulocyte-Macrophage Colony-Stimulating Factor