The C228T mutation of TERT promoter frequently occurs in bladder cancer stem cells and contributes to tumorigenesis of bladder cancer

Oncotarget. 2015 Aug 14;6(23):19542-51. doi: 10.18632/oncotarget.4295.

Abstract

Bladder cancer is one of the most common malignant tumors worldwide. Bladder cancer stem cells (BCSCs) have been isolated recently but have not been defined yet. Here we sorted BCSCs from bladder tumor tissues or normal bladder stem cells (NBBCs) from adjacent normal bladder tissues. We found that the C228T mutation (chr5, 1, 295, 228 C > T) of TERT promoter frequently occurs in BCSCs, but not exist in NBBCs. Importantly, introducing the C228T mutation in NBBCs causes TERT overexpression and transformation of bladder cancer. Restoration of the C228T mutation to T228C in BCSCs can recover the TERT expression to a basal level and abolish tumor formation. Additionally, the C228T mutation of TERT promoter triggers TERT expression leading to increased telomerase activity. TERT expression levels are consistent with clinical severity and prognosis of bladder cancer.

Keywords: C228T mutation; bladder cancer stem cells; telomerase reverse transcriptase (TERT); tumorigenesis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Biomarkers, Tumor / genetics*
  • Cell Transformation, Neoplastic / genetics
  • Cell Transformation, Neoplastic / metabolism
  • Cell Transformation, Neoplastic / pathology
  • Gene Frequency
  • Genetic Predisposition to Disease
  • Humans
  • Mice, Inbred NOD
  • Mice, SCID
  • Mutation*
  • Neoplastic Stem Cells / enzymology*
  • Phenotype
  • Prognosis
  • Promoter Regions, Genetic*
  • Risk Factors
  • Telomerase / genetics*
  • Time Factors
  • Transfection
  • Tumor Cells, Cultured
  • Urinary Bladder Neoplasms / enzymology*
  • Urinary Bladder Neoplasms / genetics*
  • Urinary Bladder Neoplasms / pathology

Substances

  • Biomarkers, Tumor
  • TERT protein, human
  • Telomerase