CaMKII plays a part in the chondrogenesis of bone marrow-derived mesenchymal stem cells

Int J Clin Exp Pathol. 2015 May 1;8(5):5981-7. eCollection 2015.

Abstract

Aims: The purpose of the study is to observe the functions of calcium/calmodulin dependent protein kinase II (CaMKII) in the induced chondrogenic differentiation of bone marrow derived mesenchymal stem cells (BMSCs).

Methods: BMSCs was in vitro isolated and cultured for induced chondrogenesis. Western blot was used to ascertain the expression of CaMKII and phosphorylated CaMKII (PCaMKII, activatory CaMKII) in chondrogenic induced BMSCs. MTT method was utilized to observe the impact of CaMKII on the proliferation of BMSCs. The generation of cartilage matrix in BMSCs cells was detected by toluidine blue staining. The levels of cartilage marker genes COL2A1, Aggrecan and SOX9 in BMSCs were gained by real-time fluorescence quantitative polymerase chain reaction (RT-QPCR). Finally, BMSCs proliferation, cartilage matrix generation and the changes of COL2A1, Aggrecan and SOX9 were surveyed after CaMKII being blocked by CaMKII inhibitor KN93.

Results: Expression of CaMKII and PCaMKII could be found in chondrogenic induced BMSCs. CaMKII had no significant influence on BMSCs proliferation, but the toluidine blue staining was obviously lighter, indicating a significant decline in the expression of COL2A1, Aggrecan and SOX9.

Conclusion: As one of the factors influencing the chondrogenic capacity of BMSCs, CaMKII does not impact on BMSCs proliferation, but it can inhibit the chondrogenic ability of BMSCs by influencing its differentiation.

Keywords: CaMKII; bone marrow derived mesenchymal stem cells (BMSCs); chondrogenesis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aggrecans / genetics
  • Aggrecans / metabolism
  • Animals
  • Bone Marrow Cells / drug effects
  • Bone Marrow Cells / enzymology*
  • Calcium-Calmodulin-Dependent Protein Kinase Type 2 / antagonists & inhibitors
  • Calcium-Calmodulin-Dependent Protein Kinase Type 2 / metabolism*
  • Cell Differentiation
  • Cell Proliferation
  • Cells, Cultured
  • Chondrocytes / drug effects
  • Chondrocytes / enzymology*
  • Chondrogenesis* / drug effects
  • Chondrogenesis* / genetics
  • Collagen Type II / genetics
  • Collagen Type II / metabolism
  • Gene Expression Regulation
  • Mesenchymal Stem Cells / drug effects
  • Mesenchymal Stem Cells / enzymology*
  • Phosphorylation
  • Protein Kinase Inhibitors / pharmacology
  • Rats
  • SOX9 Transcription Factor / genetics
  • SOX9 Transcription Factor / metabolism
  • Signal Transduction
  • Time Factors

Substances

  • Aggrecans
  • Col2a1 protein, mouse
  • Collagen Type II
  • Protein Kinase Inhibitors
  • SOX9 Transcription Factor
  • SOX9 protein, human
  • Calcium-Calmodulin-Dependent Protein Kinase Type 2