MiR-345 suppresses proliferation, migration and invasion by targeting Smad1 in human prostate cancer

J Cancer Res Clin Oncol. 2016 Jan;142(1):213-24. doi: 10.1007/s00432-015-2016-0. Epub 2015 Jul 31.

Abstract

Introduction: The roles of dysregulated microRNAs in prostate cancer metastasis are still unknown. In this study, we found that the expression of miR-345 was significantly downregulated in prostate cancer and clinical prostate cancer tissues.

Materials, methods and results: Overexpression of miR-345 in prostate cancer cells suppressed proliferation, migration and invasion. Using nude mice model, we revealed that miR-345 inhibits the growth of prostate cancer cells in vivo and in vitro. Furthermore, we identified and validated Smad1 as a direct target of miR-345. Ectopic expression of Smad1 without its 3'-UTR rescued miR-345-induced cell migration and invasion inhibition.

Conclusion: Taken together, our data suggest that miR-345 exerts a suppressive effect on prostate cancer proliferation, invasion and migration through downregulation of Smad1.

Keywords: Prostate cancer; Smad1; miR-345; microRNA.

MeSH terms

  • Aged
  • Aged, 80 and over
  • Animals
  • Apoptosis
  • Blotting, Western
  • Cell Adhesion
  • Cell Movement
  • Cell Proliferation
  • Follow-Up Studies
  • Humans
  • Immunoenzyme Techniques
  • Lymphatic Metastasis
  • Male
  • Mice
  • Mice, Nude
  • MicroRNAs / genetics*
  • Middle Aged
  • Neoplasm Grading
  • Neoplasm Staging
  • Prognosis
  • Prostatic Neoplasms / genetics*
  • Prostatic Neoplasms / mortality
  • Prostatic Neoplasms / pathology*
  • RNA, Messenger / genetics
  • Real-Time Polymerase Chain Reaction
  • Reverse Transcriptase Polymerase Chain Reaction
  • Smad1 Protein / genetics
  • Smad1 Protein / metabolism*
  • Survival Rate
  • Tumor Cells, Cultured
  • Xenograft Model Antitumor Assays

Substances

  • MIRN345 microRNA, human
  • MicroRNAs
  • RNA, Messenger
  • SMAD1 protein, human
  • Smad1 Protein