Interfacial inhibitors

Bioorg Med Chem Lett. 2015 Sep 15;25(18):3961-5. doi: 10.1016/j.bmcl.2015.07.032. Epub 2015 Jul 18.

Abstract

Targeting macromolecular interface is a general mechanism by which natural products inactivate macromolecular complexes by stabilizing normally transient intermediates. Demonstrating interfacial inhibition mechanism ultimately relies on the resolution of drug-macromolecule structures. This review focuses on medicinal drugs that trap protein-DNA complexes by binding at protein-DNA interfaces. It provides proof-of-concept and detailed structural and mechanistic examples for topoisomerase inhibitors and HIV integrase inhibitors. Additional examples of recent interfacial inhibitors for protein-DNA interfaces are provided, as well as prospects for targeting previously 'undruggable' targets including transcription, replication and chromatin remodeling complexes. References and discussion are included for interfacial inhibitors of protein-protein interfaces.

Keywords: Chemotherapy; Integrase; Natural products; Pharmacology; Topoisomerase.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, N.I.H., Intramural
  • Review

MeSH terms

  • Binding Sites / drug effects*
  • DNA / chemistry
  • DNA / metabolism
  • DNA Topoisomerases / chemistry
  • DNA Topoisomerases / metabolism
  • HIV Integrase / chemistry
  • HIV Integrase / metabolism
  • HIV Integrase Inhibitors / chemistry
  • HIV Integrase Inhibitors / pharmacology*
  • Humans
  • Macromolecular Substances / chemistry
  • Macromolecular Substances / metabolism*
  • Structure-Activity Relationship
  • Topoisomerase Inhibitors / chemistry
  • Topoisomerase Inhibitors / pharmacology*

Substances

  • HIV Integrase Inhibitors
  • Macromolecular Substances
  • Topoisomerase Inhibitors
  • DNA
  • HIV Integrase
  • DNA Topoisomerases