MGMT inactivation and clinical response in newly diagnosed GBM patients treated with Gliadel

J Clin Neurosci. 2015 Dec;22(12):1938-42. doi: 10.1016/j.jocn.2015.07.003. Epub 2015 Aug 4.

Abstract

We examined the relationship between the O(6)-methylguanine-methyltransferase (MGMT) methylation status and clinical outcomes in newly diagnosed glioblastoma multiforme (GBM) patients who were treated with Gliadel wafers (Eisai, Tokyo, Japan). MGMT promoter methylation has been associated with increased survival among patients with GBM who are treated with various alkylating agents. MGMT promoter methylation, in DNA from 122 of 160 newly diagnosed GBM patients treated with Gliadel, was determined by a quantitative methylation-specific polymerase chain reaction, and was correlated with overall survival (OS) and recurrence-free survival (RFS). The MGMT promoter was methylated in 40 (32.7%) of 122 patients. The median OS was 13.5 months (95% confidence interval [CI] 11.0-14.5) and RFS was 9.4 months (95% CI 7.8-10.2). After adjusting for age, Karnofsky performance score, extent of resection, temozolomide (TMZ) and radiation therapy (RT), the newly diagnosed GBM patients with MGMT methylation had a 15% reduced mortality risk, compared to patients with unmethylated MGMT (hazard ratio 0.85; 95% CI 0.56-1.31; p=0.46). The patients aged over 70 years with MGMT methylation had a significantly longer median OS of 13.5 months, compared to 7.6 months in patients with unmethylated MGMT (p=0.027). A significant difference was also found in older patients, with a median RFS of 13.1 versus 7.6 months for methylated and unmethylated MGMT groups, respectively (p=0.01). Methylation of the MGMT promoter in newly diagnosed GBM patients treated with Gliadel, RT and TMZ, was associated with significantly improved OS compared to the unmethylated population. In elderly patients, methylation of the MGMT promoter was associated with significantly better OS and RFS.

Keywords: Gliadel; Glioblastoma multiforme; MGMT; Methylation; Outcome; Survival.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Aged, 80 and over
  • Brain Neoplasms / genetics*
  • Brain Neoplasms / therapy
  • Carmustine / administration & dosage
  • Chemoradiotherapy
  • DNA Methylation / genetics*
  • DNA Modification Methylases / genetics*
  • DNA Repair Enzymes / genetics*
  • Dacarbazine / administration & dosage
  • Dacarbazine / analogs & derivatives
  • Decanoic Acids / administration & dosage
  • Female
  • Glioblastoma / genetics*
  • Glioblastoma / therapy
  • Humans
  • Japan
  • Karnofsky Performance Status
  • Male
  • Middle Aged
  • Polyesters / administration & dosage
  • Prognosis
  • Promoter Regions, Genetic / genetics*
  • Temozolomide
  • Tumor Suppressor Proteins / genetics*

Substances

  • Decanoic Acids
  • Polyesters
  • Tumor Suppressor Proteins
  • carmustine, poliferprosan 20 drug combination
  • Dacarbazine
  • DNA Modification Methylases
  • MGMT protein, human
  • DNA Repair Enzymes
  • Carmustine
  • Temozolomide