Paralytic activity of (des-Glu1)conotoxin GI analogs in the mouse diaphragm

Int J Pept Protein Res. 1989 Dec;34(6):455-62. doi: 10.1111/j.1399-3011.1989.tb01394.x.

Abstract

A series of 20 peptide analogs of (des-Glu1)conotoxin GI were prepared by solid phase synthesis. The peptides were tested for their abilities to inhibit contractions in the mouse-diaphragm-with-phrenic-nerve assay. (Des-Glu1)conotoxin has an IC50 of 2.7 x 10(-7) M in this assay. Results from this assay show that total loss of paralytic activity occurs when Pro is replaced by Gly, Tyr by D-Tyr, or Gly by D-Phe. In most cases loss or change in length of one of the disulfide rings eliminates paralytic activity except with compound 17, which is weakly active, IC50 = 7.0 x 10(-5) M. Replacement of the Cys1-Cys6 disulfide bond with an amide bond (compound 9) greatly lowers paralytic activity, IC50 = 3.7 x 10(-5) M.

Publication types

  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Conotoxins*
  • Diaphragm / drug effects
  • In Vitro Techniques
  • Male
  • Mice
  • Molecular Sequence Data
  • Mollusk Venoms / chemical synthesis
  • Mollusk Venoms / toxicity*
  • Neuromuscular Junction / drug effects*
  • Paralysis / chemically induced*
  • Rats
  • Structure-Activity Relationship

Substances

  • Conotoxins
  • Mollusk Venoms
  • conotoxin GI, des-Glu(1)-