Pharmacodynamics (PD) and pharmacokinetics (PK) of E7389 (eribulin, halichondrin B analog) during a phase I trial in patients with advanced solid tumors: a California Cancer Consortium trial

Cancer Chemother Pharmacol. 2015 Nov;76(5):897-907. doi: 10.1007/s00280-015-2868-7. Epub 2015 Sep 11.

Abstract

Background: The California Cancer Consortium completed a phase I trial of E7389 (eribulin mesylate), an analog of the marine natural product halichondrin B. This trial was to determine the pharmacodynamics, pharmacokinetics, and MTD of E7389 administered by bolus injection weekly for 3 weeks out of four.

Methods: This trial included a rapid titration design. Real-time pharmacokinetics were utilized to guide dose escalation. Initially, single-patient cohorts were enrolled with intra- and inter-patient dose doubling. The second phase was a standard 3 + 3 dose escalation schedule. At the MTD, a cohort of patients was enrolled for target validation studies (separate manuscript). The starting dose was 0.125 mg/m(2), and doses were doubled within and between patients in the first phase. Blood and urine sampling for E7389 pharmacokinetics was performed on doses 1 and 3 of cycle 1. Levels were determined using a LC/MS/MS assay.

Results: Forty patients were entered. Thirty-eight were evaluable for toxicity and 35 for response. The rapid escalation ended with a grade 3 elevation of alkaline phosphatase at 0.5 mg/m(2)/week. The second phase ended at 2.0 mg/m(2)/week with dose-limiting toxicities of grades 3 and 4 febrile neutropenia. Other toxicities included hypoglycemia, hypophosphatemia, and fatigue. The MTD was 1.4 mg/m(2)/week. Responses included four partial responses (lung cancer [2], urothelial [1], and melanoma [1]).

Conclusions: E7389 was well tolerated in this trial with the major toxicity being myelosuppression. PD shows that E7389 induces significant morphologic changes (bundle formation) in the microtubules of peripheral blood mononuclear cells and tumor cells in vivo. The data suggest that lower intra-tumoral levels of β-tubulin III or higher intra-tumoral levels of MAP4 may correlate with response to E7389, while lower intra-tumoral levels of stathmin may be associated with progression. PK data reveal that E7389 exhibits a tri-exponential elimination from the plasma of patients receiving a rapid i.v. infusion. At sub-toxic doses, plasma concentrations of E7389 are maintained well above the levels required for activity in vitro for >72 h.

Keywords: Eribulin; Halichondrin B analog; Pharmacodynamics; Pharmacokinetics; Phase I trial.

Publication types

  • Clinical Trial, Phase I
  • Comparative Study
  • Multicenter Study
  • Research Support, N.I.H., Extramural

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Antineoplastic Agents / adverse effects
  • Antineoplastic Agents / blood
  • Antineoplastic Agents / pharmacokinetics
  • Antineoplastic Agents / pharmacology*
  • Antineoplastic Agents / therapeutic use
  • Breast Neoplasms / blood
  • Breast Neoplasms / drug therapy
  • Carcinoma / blood
  • Carcinoma / drug therapy*
  • Dose-Response Relationship, Drug
  • Drug Monitoring
  • Febrile Neutropenia / chemically induced
  • Female
  • Furans / adverse effects
  • Furans / blood
  • Furans / pharmacokinetics
  • Furans / pharmacology*
  • Furans / therapeutic use
  • Humans
  • Injections, Intravenous
  • Ketones / adverse effects
  • Ketones / blood
  • Ketones / pharmacokinetics
  • Ketones / pharmacology*
  • Ketones / therapeutic use
  • Lung Neoplasms / blood
  • Lung Neoplasms / drug therapy
  • Lymphopenia / chemically induced
  • Male
  • Melanoma / blood
  • Melanoma / drug therapy
  • Middle Aged
  • Ovarian Neoplasms / blood
  • Ovarian Neoplasms / drug therapy
  • Salvage Therapy
  • Tubulin Modulators / adverse effects
  • Tubulin Modulators / blood
  • Tubulin Modulators / pharmacokinetics
  • Tubulin Modulators / pharmacology*
  • Tubulin Modulators / therapeutic use
  • Urologic Neoplasms / blood
  • Urologic Neoplasms / drug therapy

Substances

  • Antineoplastic Agents
  • Furans
  • Ketones
  • Tubulin Modulators
  • eribulin