Up-regulation of Toll-like Receptor 9 in Osteosarcoma

Anticancer Res. 2015 Nov;35(11):5839-43.

Abstract

Background: Increasing evidence has shown that Toll-like receptors (TLRs), key receptors in innate immunity, play a role in cancer development and progression. The present study aimed to elucidate the role of TLR expression in osteosarcoma cancer cells and patient specimens.

Materials and methods: We investigated the expression of all of human TLRs in osteosarcoma MG-63 cells by real-time quantitative reverse transcription polymerase chain reaction. We then further explored whether the up-regulation of TLR9 expression is common in patients with osteosarcoma by examining TLR9 protein levels in 80 osteosarcoma specimens and 28 normal controls by immunohistochemistry.

Results: We found that among TLR family members, TLR9 was predominately expressed in osteosarcoma cells, and up-regulation of TLR9 expression was found in 72 out of 80 (90%) patients with osteosarcoma but in none of 28 normal controls. Furthermore, high expression of TLR9 appeared to be associated with osteosarcoma progression.

Conclusion: TLR9 is up-regulated in the majority of osteosarcomas, which appears to play an important role in osteosarcoma development and progression. Therefore, TLR9 may serve as a novel therapeutic target for human osteosarcoma therapy.

Keywords: MG-63 cells; Osteosarcoma; TLR9; innate immunity; toll-like receptor.

Publication types

  • Comparative Study
  • Research Support, N.I.H., Extramural

MeSH terms

  • Adult
  • Bone Neoplasms / genetics
  • Bone Neoplasms / metabolism*
  • Bone Neoplasms / pathology
  • Case-Control Studies
  • Disease Progression
  • Female
  • Follow-Up Studies
  • Humans
  • Immunoenzyme Techniques
  • Lymphatic Metastasis
  • Male
  • Neoplasm Grading
  • Osteosarcoma / genetics
  • Osteosarcoma / metabolism*
  • Osteosarcoma / secondary
  • Prognosis
  • RNA, Messenger / genetics
  • Real-Time Polymerase Chain Reaction
  • Reverse Transcriptase Polymerase Chain Reaction
  • Toll-Like Receptors / genetics
  • Toll-Like Receptors / metabolism*
  • Tumor Cells, Cultured
  • Up-Regulation
  • Young Adult

Substances

  • RNA, Messenger
  • Toll-Like Receptors