Low MiR-149 expression is associated with unfavorable prognosis and enhanced Akt/mTOR signaling in glioma

Int J Clin Exp Pathol. 2015 Sep 1;8(9):11178-84. eCollection 2015.

Abstract

MicroRNAs (miRs) play critical roles in the progression of glioma. Previous in vitro studies have described the anti-tumor role of miR-149 in cancer cells including glioma. In this study, we aimed to investigate whether miR-149 is associated with the prognosis of glioma patients. A total of 163 glioma patients who underwent tumor resection were included in our follow-up study. We found that the miR-149 expression was significantly lower in tumor tissues compared with that in normal tissues (P<0.05). Kaplan-Meier and analysis showed that the miR-149 expression status was significantly associated with the survival duration (logrank test, P<0.001), and multivariate Cox regression revealed that patients with low miR-149 expression were exposed to a 1.825 fold higher death risk (HR=1.825, 95% CI=1.031-3.229, P=0.039) compared with those with high miR-149 expression. Further study showed that Akt/mTOR signaling was hyperactive in low miR-149 expressing tissues. Our study thus demonstrates that miR-149 expression in glioma tissues is critically associated with the prognosis of patients, suggesting its potential clinical significance.

Keywords: Akt; Glioma; mTOR; miR-149; prognosis.

MeSH terms

  • Chi-Square Distribution
  • Down-Regulation
  • Female
  • Gene Expression Regulation, Neoplastic
  • Glioma / enzymology*
  • Glioma / genetics*
  • Glioma / mortality
  • Glioma / pathology
  • Glioma / surgery
  • Humans
  • Kaplan-Meier Estimate
  • Male
  • MicroRNAs / genetics*
  • Middle Aged
  • Multivariate Analysis
  • Phosphorylation
  • Prognosis
  • Proportional Hazards Models
  • Proto-Oncogene Proteins c-akt / analysis*
  • Risk Assessment
  • Risk Factors
  • Signal Transduction*
  • TOR Serine-Threonine Kinases / analysis*
  • Time Factors

Substances

  • MIRN149 microRNA, human
  • MicroRNAs
  • MTOR protein, human
  • Proto-Oncogene Proteins c-akt
  • TOR Serine-Threonine Kinases