High-content analysis of α-synuclein aggregation and cell death in a cellular model of Parkinson's disease

J Neurosci Methods. 2016 Mar 1:261:117-27. doi: 10.1016/j.jneumeth.2015.11.009. Epub 2015 Nov 24.

Abstract

Background: Alpha-synuclein (α-SYN) aggregates represent a key feature of Parkinson's disease, but the exact relationship between α-SYN aggregation and neurodegeneration remains incompletely understood. Therefore, the availability of a cellular assay that allows medium-throughput analysis of α-SYN-linked pathology will be of great value for studying the aggregation process and for advancing α-SYN-based therapies.

New method: Here we describe a high-content neuronal cell assay that simultaneously measures oxidative stress-induced α-SYN aggregation and apoptosis.

Results: We optimized an automated and reproducible assay to quantify both α-SYN aggregation and cell death in human SH-SY5Y neuroblastoma cells.

Comparison with existing methods: Quantification of α-SYN aggregates in cells has typically relied on manual imaging and counting or cell-free assays, which are time consuming and do not allow a concurrent analysis of cell viability. Our high-content analysis method for quantification of α-SYN aggregation allows simultaneous measurements of multiple cell parameters at a single-cell level in a fast, objective and automated manner.

Conclusions: The presented analysis approach offers a rapid, objective and multiparametric approach for the screening of compounds and genes that might alter α-SYN aggregation and/or toxicity.

Keywords: Aggregation; Alpha-synuclein; Cell death; High-content analysis; Parkinson's disease.

Publication types

  • Research Support, Non-U.S. Gov't
  • Validation Study

MeSH terms

  • Apoptosis*
  • Benzothiazoles
  • Blotting, Western
  • Cell Count
  • Cell Culture Techniques / methods
  • Cell Line, Tumor
  • Cell Survival
  • Genetic Vectors
  • Humans
  • Image Processing, Computer-Assisted / methods*
  • Immunohistochemistry / methods*
  • Indoles
  • Lentivirus / genetics
  • Microscopy, Fluorescence / methods
  • Oxidative Stress
  • Parkinsonian Disorders / metabolism*
  • Parkinsonian Disorders / pathology*
  • Protein Aggregation, Pathological*
  • Protein Multimerization
  • Software
  • Thiazoles
  • alpha-Synuclein / chemistry*
  • alpha-Synuclein / genetics
  • alpha-Synuclein / metabolism

Substances

  • Benzothiazoles
  • Indoles
  • SNCA protein, human
  • Thiazoles
  • alpha-Synuclein
  • thioflavin T
  • DAPI