Expression Profiling after Prolonged Experimental Febrile Seizures in Mice Suggests Structural Remodeling in the Hippocampus

PLoS One. 2015 Dec 18;10(12):e0145247. doi: 10.1371/journal.pone.0145247. eCollection 2015.

Abstract

Febrile seizures are the most prevalent type of seizures among children up to 5 years of age (2-4% of Western-European children). Complex febrile seizures are associated with an increased risk to develop temporal lobe epilepsy. To investigate short- and long-term effects of experimental febrile seizures (eFS), we induced eFS in highly febrile convulsion-susceptible C57BL/6J mice at post-natal day 10 by exposure to hyperthermia (HT) and compared them to normotherm-exposed (NT) mice. We detected structural re-organization in the hippocampus 14 days after eFS. To identify molecular candidates, which entrain this structural re-organization, we investigated temporal changes in mRNA expression profiles eFS 1 hour to 56 days after eFS. We identified 931 regulated genes and profiled several candidates using in situ hybridization and histology at 3 and 14 days after eFS. This is the first study to report genome-wide transcriptome analysis after eFS in mice. We identify temporal regulation of multiple processes, such as stress-, immune- and inflammatory responses, glia activation, glutamate-glutamine cycle and myelination. Identification of the short- and long-term changes after eFS is important to elucidate the mechanisms contributing to epileptogenesis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase / genetics
  • 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase / metabolism
  • Animals
  • CA1 Region, Hippocampal / metabolism*
  • CA1 Region, Hippocampal / pathology
  • CA3 Region, Hippocampal / metabolism*
  • CA3 Region, Hippocampal / pathology
  • Epilepsy, Temporal Lobe / metabolism
  • Epilepsy, Temporal Lobe / pathology
  • Female
  • Gene Expression Profiling
  • Gene Expression Regulation
  • Gene Ontology
  • Heat-Shock Response
  • Male
  • Mice, Inbred C57BL
  • Neurofilament Proteins / genetics
  • Neurofilament Proteins / metabolism
  • Seizures, Febrile / metabolism*
  • Seizures, Febrile / pathology
  • Transcriptome*
  • Up-Regulation

Substances

  • Neurofilament Proteins
  • 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase
  • Cnp protein, mouse

Grants and funding

This work was supported by the National Epilepsy Fund of the Netherlands, grant number: NEF 10-17 and 03-12, (https://www.epilepsie.nl), and the Epilepsies of Childhood Foundation, grant number: N/A, website: N/A. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.