Abstract
We show here that the Plasmodium falciparum isolate FCR3 does not express the ring-infected erythrocyte surface antigen (RESA). This is because the 5' end of the RESA gene has been inverted and partly deleted and a telomere has been added to it. We propose a model to explain these events.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Amino Acid Sequence
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Animals
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Antigens, Protozoan / genetics*
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Antigens, Surface / genetics*
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Antigens, Surface / immunology
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Base Sequence
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Chromosome Deletion
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Chromosome Inversion
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Chromosome Mapping
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Gene Amplification
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Immunoblotting
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Molecular Sequence Data
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Mutation
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Plasmodium falciparum / genetics*
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Plasmodium falciparum / immunology
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Protozoan Proteins*
Substances
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Antigens, Protozoan
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Antigens, Surface
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Protozoan Proteins
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ring-infected erythrocyte surface antigen (RESA), Plasmodium falciparum