Tumor Microenvironment-Mediated Construction and Deconstruction of Extracellular Drug-Delivery Depots

Nano Lett. 2016 Feb 10;16(2):1118-26. doi: 10.1021/acs.nanolett.5b04343. Epub 2016 Jan 19.

Abstract

Protein therapy has been considered the most direct and safe approach to treat cancer. Targeting delivery of extracellularly active protein without internalization barriers, such as membrane permeation and endosome escape, is efficient and holds vast promise for anticancer treatment. Herein, we describe a "transformable" core-shell based nanocarrier (designated CS-NG), which can enzymatically assemble into microsized extracellular depots at the tumor site with assistance of hyaluronidase (HAase), an overexpressed enzyme at the tumor microenvironment. Equipped with an acid-degradable modality, the resulting CS-NG can substantially release combinational anticancer drugs-tumor necrosis factor (TNF)-related apoptosis inducing ligand (TRAIL) and antiangiogenic cilengitide toward the membrane of cancer cells and endothelial cells at the acidic tumor microenvironment, respectively. Enhanced cytotoxicity on MDA-MB-231 cells and improved antitumor efficacy were observed using CS-NG, which was attributed to the inhibition of cellular internalization and prolonged retention time in vivo.

Keywords: Drug delivery; antiangiogenesis; apoptosis; stimuli-responsive; tumor microenvironment.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Angiogenesis Inhibitors / administration & dosage
  • Angiogenesis Inhibitors / chemistry
  • Animals
  • Antineoplastic Combined Chemotherapy Protocols / administration & dosage
  • Antineoplastic Combined Chemotherapy Protocols / chemistry
  • Apoptosis / drug effects
  • Breast Neoplasms / drug therapy*
  • Breast Neoplasms / genetics
  • Cell Line, Tumor
  • Cell Membrane Permeability / drug effects
  • Drug Delivery Systems*
  • Endosomes / drug effects
  • Gene Transfer Techniques*
  • Humans
  • Hyaluronoglucosaminidase / biosynthesis
  • Hyaluronoglucosaminidase / chemistry
  • Mice
  • Snake Venoms / administration & dosage*
  • Snake Venoms / chemistry
  • TNF-Related Apoptosis-Inducing Ligand / administration & dosage*
  • Tumor Microenvironment / drug effects
  • Xenograft Model Antitumor Assays

Substances

  • Angiogenesis Inhibitors
  • Snake Venoms
  • TNF-Related Apoptosis-Inducing Ligand
  • Cilengitide
  • Hyaluronoglucosaminidase