Chlorogenic acid ameliorates intestinal mitochondrial injury by increasing antioxidant effects and activity of respiratory complexes

Biosci Biotechnol Biochem. 2016 May;80(5):962-71. doi: 10.1080/09168451.2015.1127130. Epub 2016 Jan 29.

Abstract

Dietary polyphenols are thought to be beneficial for human health by acting as antioxidants. Chlorogenic acid (CGA) is abundant in plant-based foods as an ester of caffeic acid and quinic acid. In this study, we investigated the effects of CGA on mitochondrial protection. Our results demonstrated that pretreatment with CGA ameliorated the intestinal mitochondrial injury induced by H2O2; membrane potential was increased, mitochondrial swelling, levels of reactive oxygen species, contents of 8-hydroxy-2-deoxyguanosine, and cytochrome c released were decreased. The beneficial effects of CGA were accompanied by an increase in antioxidant and respiratory-chain complex I, IV, and V activities. In trinitrobenzene-sulfonic acid-induced colitic rats indicated that CGA supplementation improved mitochondria ultrastructure and decreased mitochondrial injury. Our results suggest a promising role for CGA as a mitochondria-targeted antioxidant in combating intestinal oxidative injury. Daily intake of diets containing CGA, such as coffee and honeysuckle, may be useful for prevention of intestinal diseases.

Keywords: chlorogenic acid; intestinal injury; mitochondria; respiratory chain complex.

MeSH terms

  • Animals
  • Antioxidants / pharmacology*
  • Chlorogenic Acid / pharmacology*
  • Colitis / chemically induced
  • Colitis / drug therapy*
  • Colitis / metabolism
  • Colitis / pathology
  • Cytochromes c / metabolism*
  • Electron Transport Complex I / metabolism*
  • Electron Transport Complex IV / metabolism*
  • Enzyme Activation
  • Female
  • Glutathione / metabolism
  • Hydrogen Peroxide / antagonists & inhibitors
  • Hydrogen Peroxide / pharmacology
  • Membrane Potential, Mitochondrial / drug effects
  • Mitochondria / drug effects*
  • Mitochondria / metabolism
  • Mitochondria / pathology
  • Mitochondrial Swelling / drug effects
  • Oxidation-Reduction
  • Oxidative Stress / drug effects
  • Rats
  • Rats, Sprague-Dawley
  • Reactive Oxygen Species / antagonists & inhibitors
  • Reactive Oxygen Species / metabolism
  • Trinitrobenzenesulfonic Acid

Substances

  • Antioxidants
  • Reactive Oxygen Species
  • Chlorogenic Acid
  • Trinitrobenzenesulfonic Acid
  • Cytochromes c
  • Hydrogen Peroxide
  • Electron Transport Complex IV
  • Electron Transport Complex I
  • Glutathione