Allograft inflammatory factor-1 in the pathogenesis of bleomycin-induced acute lung injury

Biosci Trends. 2016 Feb;10(1):47-53. doi: 10.5582/bst.2016.01027. Epub 2016 Feb 25.

Abstract

Allograft inflammatory factor-1 (AIF-1) is a protein expressed by macrophages infiltrating the area around the coronary arteries of rats with an ectopic cardiac allograft. Some studies have shown that expression of AIF-1 increased in a mouse model of trinitrobenzene sulfonic acid-induced acute colitis and in acute cellular rejection of human cardiac allografts. These results suggest that AIF-1 is related to acute inflammation. The current study used bleomycin-induced acute lung injury to analyze the expression of AIF-1 and to examine its function in acute lung injury. Results showed that AIF-1 was significantly expressed in lung macrophages and increased in bronchoalveolar lavage fluid from mice with bleomycin-induced acute lung injury in comparison to control mice. Recombinant AIF-1 increased the production of IL-6 and TNF-α from RAW264.7 (a mouse macrophage cell line) and primary lung fibroblasts, and it also increased the production of KC (CXCL1) from lung fibroblasts. These results suggest that AIF-1 plays an important role in the mechanism underlying acute lung injury.

MeSH terms

  • Acute Lung Injury / chemically induced*
  • Acute Lung Injury / complications
  • Acute Lung Injury / metabolism*
  • Acute Lung Injury / pathology
  • Animals
  • Bleomycin
  • Bronchoalveolar Lavage Fluid / cytology
  • Calcium-Binding Proteins / metabolism*
  • Chemokine CXCL1 / metabolism
  • Fibroblasts / metabolism
  • Fibroblasts / pathology
  • Humans
  • Inflammation / complications
  • Inflammation / pathology
  • Interleukin-6 / metabolism
  • Lung / pathology
  • Macrophages, Alveolar / metabolism
  • Macrophages, Alveolar / pathology
  • Mice
  • Mice, Inbred C57BL
  • Microfilament Proteins / metabolism*
  • RAW 264.7 Cells
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Aif1 protein, mouse
  • Calcium-Binding Proteins
  • Chemokine CXCL1
  • Interleukin-6
  • Microfilament Proteins
  • Tumor Necrosis Factor-alpha
  • Bleomycin