ADAMTS13-mediated thrombolysis of t-PA-resistant occlusions in ischemic stroke in mice

Blood. 2016 May 12;127(19):2337-45. doi: 10.1182/blood-2015-08-662650. Epub 2016 Feb 29.

Abstract

Rapid vascular recanalization forms the basis for successful treatment of cerebral ischemia. Currently, tissue plasminogen activator (t-PA) is the only approved thrombolytic drug for ischemic stroke. However, t-PA does not always result in efficient thrombus dissolution and subsequent blood vessel recanalization. To better understand thrombus composition, we analyzed thrombi retrieved from ischemic stroke patients and found a distinct presence of von Willebrand factor (VWF) in various samples. Thrombi contained on average 20.3% ± 10.1% VWF, and this was inversely correlated with thrombus red blood cell content. We hypothesized that ADAMTS13 can exert a thrombolytic effect in VWF-containing thrombi in the setting of stroke. To test this, we generated occlusive VWF-rich thrombi in the middle cerebral artery (MCA) of mice. Infusion of t-PA did not dissolve these MCA occlusions. Interestingly, administration of ADAMTS13 5 minutes after occlusion dose-dependently dissolved these t-PA-resistant thrombi resulting in fast restoration of MCA patency and consequently reduced cerebral infarct sizes (P < .005). Delayed ADAMTS13 administration 60 minutes after occlusion was still effective but to a lesser extent (P < .05). These data show for the first time a potent thrombolytic activity of ADAMTS13 in the setting of stroke, which might become useful in treatment of acute ischemic stroke.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • ADAMTS13 Protein / genetics
  • ADAMTS13 Protein / metabolism
  • ADAMTS13 Protein / pharmacology*
  • Animals
  • Brain Ischemia / drug therapy*
  • Brain Ischemia / genetics
  • Brain Ischemia / metabolism
  • Disease Models, Animal
  • Drug Resistance / drug effects*
  • Drug Resistance / genetics
  • Female
  • Male
  • Mice
  • Mice, Knockout
  • Stroke / drug therapy*
  • Stroke / genetics
  • Stroke / metabolism
  • Thrombolytic Therapy*
  • Thrombosis / drug therapy*
  • Thrombosis / genetics
  • Thrombosis / metabolism
  • Tissue Plasminogen Activator / pharmacology*

Substances

  • Tissue Plasminogen Activator
  • ADAMTS13 protein, mouse
  • ADAMTS13 Protein