Myomir dysregulation and reactive oxygen species in aged human satellite cells

Biochem Biophys Res Commun. 2016 Apr 29;473(2):462-70. doi: 10.1016/j.bbrc.2016.03.030. Epub 2016 Mar 11.

Abstract

Satellite cells that reside on the myofibre surface are crucial for the muscle homeostasis and regeneration. Aging goes along with a less effective regeneration of skeletal muscle tissue mainly due to the decreased myogenic capability of satellite cells. This phenomenon impedes proper maintenance and contributes to the age-associated decline in muscle mass, known as sarcopenia. The myogenic potential impairment does not depend on a reduced myogenic cell number, but mainly on their difficulty to complete a differentiation program. The unbalanced production of reactive oxygen species in elderly people could be responsible for skeletal muscle impairments. microRNAs are conserved post-transcriptional regulators implicated in numerous biological processes including adult myogenesis. Here, we measure the ROS level and analyze myomiR (miR-1, miR-133b and miR-206) expression in human myogenic precursors obtained from Vastus lateralis of elderly and young subjects to provide the molecular signature responsible for the differentiation impairment of elderly activated satellite cells.

Keywords: Aging; Human satellite cells; ROS; Sarcopenia; myomiRNAs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Aged, 80 and over
  • Aging*
  • Cells, Cultured
  • Gene Expression Regulation*
  • Humans
  • Male
  • MicroRNAs / genetics*
  • Muscle Development*
  • Reactive Oxygen Species / metabolism*
  • Sarcopenia / genetics
  • Sarcopenia / metabolism
  • Satellite Cells, Skeletal Muscle / cytology
  • Satellite Cells, Skeletal Muscle / metabolism*
  • Young Adult

Substances

  • MicroRNAs
  • Reactive Oxygen Species