Inhibition of IL-1R1/MyD88 signalling promotes mesenchymal stem cell-driven tissue regeneration

Nat Commun. 2016 Mar 22:7:11051. doi: 10.1038/ncomms11051.

Abstract

Tissue injury and the healing response lead to the release of endogenous danger signals including Toll-like receptor (TLR) and interleukin-1 receptor, type 1 (IL-1R1) ligands, which modulate the immune microenvironment. Because TLRs and IL-1R1 have been shown to influence the repair process of various tissues, we explored their role during bone regeneration, seeking to design regenerative strategies integrating a control of their signalling. Here we show that IL-1R1/MyD88 signalling negatively regulates bone regeneration, in the mouse. Furthermore, IL-1β which is released at the bone injury site, inhibits the regenerative capacities of mesenchymal stem cells (MSCs). Mechanistically, IL-1R1/MyD88 signalling impairs MSC proliferation, migration and differentiation by inhibiting the Akt/GSK-3β/β-catenin pathway. Lastly, as a proof of concept, we engineer a MSC delivery system integrating inhibitors of IL-1R1/MyD88 signalling. Using this strategy, we considerably improve MSC-based bone regeneration in the mouse, demonstrating that this approach may be useful in regenerative medicine applications.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bone Regeneration / genetics*
  • Bone Regeneration / immunology
  • Cell Differentiation
  • Cell Movement
  • Cell Proliferation
  • Chondrocytes
  • Cytokines / immunology
  • Glycogen Synthase Kinase 3 / metabolism
  • Glycogen Synthase Kinase 3 beta
  • Interleukin-1beta / immunology*
  • Macrophages / immunology
  • Mesenchymal Stem Cells / immunology*
  • Mesenchymal Stem Cells / metabolism
  • Mesenchymal Stem Cells / physiology
  • Mice
  • Mice, Knockout
  • Monocytes / immunology
  • Myeloid Differentiation Factor 88 / genetics*
  • Osteoblasts / metabolism*
  • Osteoblasts / physiology
  • Proto-Oncogene Proteins c-akt / metabolism
  • Receptors, Interleukin-1 Type I / genetics*
  • Regeneration / genetics
  • Regeneration / immunology
  • Signal Transduction
  • Skull / diagnostic imaging
  • Skull / injuries*
  • Toll-Like Receptors / immunology
  • Wnt Signaling Pathway
  • X-Ray Microtomography
  • beta Catenin / metabolism

Substances

  • Cytokines
  • IL1B protein, mouse
  • IL1R1 protein, mouse
  • Interleukin-1beta
  • Myd88 protein, mouse
  • Myeloid Differentiation Factor 88
  • Receptors, Interleukin-1 Type I
  • Toll-Like Receptors
  • beta Catenin
  • Glycogen Synthase Kinase 3 beta
  • Gsk3b protein, mouse
  • Proto-Oncogene Proteins c-akt
  • Glycogen Synthase Kinase 3