Interleukin-22 promotes papillary thyroid cancer cell migration and invasion through microRNA-595/Sox17 axis

Tumour Biol. 2016 Sep;37(9):11753-11762. doi: 10.1007/s13277-016-5030-1. Epub 2016 Mar 29.

Abstract

Interleukin-22 (IL-22) is an inflammatory cytokine mainly produced by activated Th17 and Th22 cells. The data presented here demonstrate that IL-22 induced the migration and invasion of papillary thyroid cancer (PTC) cells. MicroRNA expression analysis and functional studies indicated that IL-22-mediated migration and invasion is positively regulated by miR-595. Further mechanistic studies revealed that sex-determining region Y-box 17 (Sox17) is directly targeted by miR-595. We then demonstrated that IL-22 regulated migration and invasion of PTC cells via inhibiting Sox17 expression. Interestingly, in PTC cell lines and PTC tissues, expression of IL-22 and miR-595 was upregulated and Sox17 downregulated compared with normal thyroid, and their expression levels were closely correlated. Taken together, this present study suggests that IL-22 stimulation enhances the migration and invasion of PTC cells by regulating miR-595 and its target Sox17.

Keywords: Interleukin-22; Invasion; Migration; Papillary thyroid cancer; Sox17; miR-595.

MeSH terms

  • Carcinoma / pathology*
  • Carcinoma, Papillary
  • Cell Line, Tumor
  • Cell Movement / drug effects
  • Humans
  • Interleukin-22
  • Interleukins / pharmacology*
  • MicroRNAs / physiology*
  • Neoplasm Invasiveness
  • SOXF Transcription Factors / genetics
  • SOXF Transcription Factors / physiology*
  • Thyroid Cancer, Papillary
  • Thyroid Neoplasms / pathology*

Substances

  • Interleukins
  • MicroRNAs
  • SOX17 protein, human
  • SOXF Transcription Factors
  • microRNA595 microRNA, human