Geraniin ameliorates cisplatin-induced nephrotoxicity in mice

Free Radic Res. 2016 Aug;50(8):813-9. doi: 10.3109/10715762.2016.1173206. Epub 2016 May 12.

Abstract

Geraniin, an active compound isolated from Geranium sibiricum, has been reported to have antioxidant and anti-inflammatory activities. The aim of this study was to investigate the protective effects of geraniin against cisplatin (CP)-induced kidney injury in mice. Geraniin was administrated for three consecutive days following CP (20 mg/kg) injection. The results showed that geraniin inhibited CP-induced kidney histopathologic changes, MDA, inflammatory cytokines tumor necrosis factor-α (TNF-α) and interleukin-1β (IL-1β) production in kidney tissues. Geraniin also inhibited CP-induced blood urea nitrogen (BUN) and creatinine production. Meanwhile, the activities of superoxide dismutase (SOD) and glutathione peroxidase (GSH-PX) decreased by CP were reversed by the treatment of geraniin. In addition, geraniin significantly inhibited CP-induced NF-κB activation in kidney tissues. Treatment of geraniin dosedependently upregulated the expression of Nrf2 and HO-1. The anticancer effects of CP were not affected by the treatment of geraniin. In conclusion, these results indicated that geraniin protected against CP-induced nephrotoxicity by inhibiting oxidative stress and inflammatory response.

Keywords: Geraniin; NF-κB; cisplatin; nephrotoxicity.

MeSH terms

  • Animals
  • Cisplatin / adverse effects*
  • Disease Models, Animal
  • Glucosides / administration & dosage
  • Glucosides / therapeutic use*
  • Hydrolyzable Tannins / administration & dosage
  • Hydrolyzable Tannins / therapeutic use*
  • Kidney / pathology*
  • Male
  • Mice
  • Mice, Inbred BALB C
  • NF-kappa B / metabolism*
  • Renal Insufficiency / chemically induced*
  • Renal Insufficiency / pathology

Substances

  • Glucosides
  • Hydrolyzable Tannins
  • NF-kappa B
  • Geraniin
  • Cisplatin