Crosstalk between TGF-β signaling and miRNAs in breast cancer metastasis

Tumour Biol. 2016 Aug;37(8):10011-9. doi: 10.1007/s13277-016-5060-8. Epub 2016 May 6.

Abstract

Transforming growth factor-β (TGF-β) signaling pathway is a key regulator of various cancer biologies, including cancer cell migration, invasion, angiogenesis, proliferation, as well as apoptosis, and it is one of indispensable signaling pathways during cancer metastasis. TGF-β signaling pathway can regulate and be regulated by a series of molecular and signaling pathways where microRNAs (miRNAs) seem to play important roles. miRNAs are small non-coding RNAs that can regulate expressions of their target genes. Emerging evidence suggest that miRNAs participate in various biological and pathologic processes such as cancer cells apoptosis, proliferation, invasion, migration, and metastasis by influencing multiple signaling pathways. In this article, we focus on the interaction between miRNAs and TGF-β in breast cancer (BC) metastasis through modulating invasion-metastasis-related factors, including epithelial-to-mesenchymal transition (EMT), cancer stem cells (CSCs), matrix metalloproteinase (MMP), tissue inhibitors of MMPs (TIMPs), cell adhesion molecules (CAMs), and tumor microenvironment (TME). Through a clear understanding of the complicated links between TGF-β pathway and miRNAs, it may provide a novel and safer therapeutic target to prevent BC metastasis.

Keywords: Breast cancer; Metastasis; TGF-β pathway; Therapy; miRNAs.

Publication types

  • Review

MeSH terms

  • Antineoplastic Agents / pharmacology
  • Apoptosis
  • Breast Neoplasms / drug therapy
  • Breast Neoplasms / genetics
  • Breast Neoplasms / pathology*
  • Cell Line, Tumor
  • Cell Movement / genetics
  • Drug Resistance, Neoplasm
  • Epithelial-Mesenchymal Transition / genetics
  • Female
  • Gene Expression Regulation, Neoplastic / drug effects
  • Humans
  • Matrix Metalloproteinases / physiology
  • MicroRNAs / genetics*
  • Models, Biological
  • Neoplasm Metastasis
  • Neoplasm Proteins / genetics*
  • Neoplasm Proteins / physiology
  • Signal Transduction / physiology*
  • Tissue Inhibitor of Metalloproteinases / physiology
  • Transforming Growth Factor beta / physiology*
  • Tumor Microenvironment

Substances

  • Antineoplastic Agents
  • MicroRNAs
  • Neoplasm Proteins
  • Tissue Inhibitor of Metalloproteinases
  • Transforming Growth Factor beta
  • Matrix Metalloproteinases