Cingulate Cortical Thickness and Dopamine Transporter ( DAT1) Genotype in Children and Adolescents With ADHD

J Atten Disord. 2018 May;22(7):651-660. doi: 10.1177/1087054716647483. Epub 2016 May 9.

Abstract

Objective: This study aimed to examine the influence of dopamine transporter gene ( DAT1) 3'UTR genotype on cingulate cortical thickness in a large sample of children and adolescents with ADHD.

Method: Brain MRIs were acquired from 46 ADHD patients with homozygosity for the 10-repeat allele and 52 ADHD patients with a single copy or no copy of the allele. The cingulate cortex of each MRI scan was automatically parceled into sulci and gyri as well as into Brodmann areas (BA).

Results: There were no group differences in age, gender, full-scale intelligence quotient, symptom severity, treatment status, comorbidity, or mean overall cortical thickness. Sulcus/gyrus- and BA-based analyses revealed that patients homozygous for the 10-repeat allele showed significantly greater thickness in right cingulate gyrus and right BA 24 compared with 9-repeat carriers.

Conclusion: These findings suggest that thickness of cingulate cortex is influenced by the presence of the 10-repeat allele in ADHD.

Keywords: ADHD; DAT1; cingulate cortex; cortical thickness.

MeSH terms

  • Adolescent
  • Alleles
  • Attention Deficit Disorder with Hyperactivity / diagnosis
  • Attention Deficit Disorder with Hyperactivity / genetics*
  • Attention Deficit Disorder with Hyperactivity / pathology
  • Child
  • Dopamine Plasma Membrane Transport Proteins / genetics*
  • Female
  • Genotype
  • Gyrus Cinguli / pathology*
  • Homozygote
  • Humans
  • Magnetic Resonance Imaging
  • Male
  • Polymorphism, Genetic / genetics*

Substances

  • Dopamine Plasma Membrane Transport Proteins
  • SLC6A3 protein, human