Cytotoxic chemotherapy and the evolution of cellular and viral resistance to antiretroviral therapy in HIV- infected individuals with lymphoma

HIV Clin Trials. 2016 Sep;17(5):197-203. doi: 10.1080/15284336.2016.1210719. Epub 2016 Jul 25.

Abstract

Background: The use of combination antiretroviral therapy (cART) and cytotoxic chemotherapy for HIV-associated lymphoma runs the risks of inducing HIV drug resistance. This study examined two possible mechanisms: altered expression of membrane drug transporter protein (MTP) and acquisition of mutations in pro-viral DNA.

Methods: Expression levels of MTP and pro-viral DNA resistance mutation analysis were performed on peripheral blood mononuclear cells (PBMC) before, during, and after chemotherapy.

Results: Twenty nine patients completed the three time point estimations. There were no significant variations before, during, and after chemotherapy in the expression of four MTPs: ABCB1, ABCC1, ABCC2, and SLCO3A1 (OATP3A1). Pro-viral DNA sequencing revealed that only one patient developed a new nucleos/tide reverse transcriptase inhibitor-associated mutation (184V) during the course of the study, giving a mutation rate of 0.0027 per person per year.

Conclusions: In conclusion, concomitant administration of cytotoxic chemotherapy and cART does not induce expression of MTP. Furthermore, no significant changes in viral resistance were observed pre- and post-chemotherapy, suggesting mutagenic cytotoxic chemotherapy seems not to induce mutations in HIV pro-viral DNA.

Keywords: Antiretroviral therapy; Chemotherapy; HIV; Lymphoma.

MeSH terms

  • Adult
  • Aged
  • Anti-HIV Agents / pharmacology*
  • Anti-HIV Agents / therapeutic use
  • Antineoplastic Agents / pharmacology*
  • Antineoplastic Agents / therapeutic use
  • Antiretroviral Therapy, Highly Active
  • CD4 Lymphocyte Count
  • Case-Control Studies
  • Drug Resistance, Neoplasm* / genetics
  • Drug Resistance, Viral*
  • Female
  • Gene Expression
  • HIV Infections / complications*
  • HIV Infections / drug therapy
  • HIV Infections / genetics
  • HIV Infections / virology*
  • HIV-1 / drug effects
  • HIV-1 / genetics
  • Humans
  • Lymphoma / drug therapy
  • Lymphoma / etiology*
  • Male
  • Membrane Transport Proteins / genetics
  • Membrane Transport Proteins / metabolism
  • Middle Aged
  • Multidrug Resistance-Associated Protein 2
  • Mutation
  • RNA, Viral
  • Viral Load

Substances

  • ABCC2 protein, human
  • Anti-HIV Agents
  • Antineoplastic Agents
  • Membrane Transport Proteins
  • Multidrug Resistance-Associated Protein 2
  • RNA, Viral