Weight Loss, Satiety, and the Postprandial Gut Hormone Response After Esophagectomy: A Prospective Study

Ann Surg. 2017 Jul;266(1):82-90. doi: 10.1097/SLA.0000000000001918.

Abstract

Objective: To prospectively characterize changes in body weight, satiety, and postprandial gut hormone profiles following esophagectomy.

Background: With improved oncologic outcomes in esophageal cancer, there is an increasing focus on functional status and health-related quality of life in survivorship. Early satiety and weight loss are common after esophagectomy, but the pathophysiology of these phenomena remains poorly understood.

Methods: In this prospective study, consecutive patients undergoing esophagectomy with gastric conduit reconstruction were studied preoperatively and at 10 days, 6 weeks, and 3 months postoperatively. Glucagon-like peptide 1 (GLP-1) immunoreactivity of plasma collected immediately before and at 15, 30, 60, 90, 120, 150, and 180 minutes after a standardized 400-kcal mixed meal was determined. Gastrointestinal symptom scores were computed using European Organization for Research and Treatment of Cancer questionnaires.

Results: Body weight loss at 6 weeks and 3 months postoperatively among 13 patients undergoing esophagectomy was 11.1 ± 2.3% (P < 0.001) and 16.3 ± 2.2% (P < 0.0001), respectively. Early satiety (P = 0.043), gastrointestinal pain and discomfort (P = 0.01), altered taste (P= 0.006), and diarrhea (P= 0.038) scores increased at 3 months postoperatively. Area under the curve for the satiety gut hormone GLP-1 was significantly increased from 10 days postoperatively (2.4 ± 0.2-fold increase, P < 0.01), and GLP-1 peak increased 3.8 ± 0.6-, 4.7 ± 0.8-, and 4.4 ± 0.5-fold at 10 days, 6 weeks, and 3 months postoperatively (all P < 0.0001). Three months postoperatively, GLP-1 area under the curve was associated with early satiety (P = 0.0002, R = 0.74), eating symptoms (P = 0.007, R = 0.54), and trouble enjoying meals (P = 0.0004, R = 0.73).

Conclusions: After esophagectomy, patients demonstrate an exaggerated postprandial satiety gut hormone response, which may mediate postoperative changes in satiety, body weight, and gastrointestinal quality of life.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Blood Glucose / metabolism
  • Esophageal Neoplasms / surgery*
  • Esophagectomy / adverse effects*
  • Female
  • Gastrointestinal Diseases / etiology
  • Glucagon-Like Peptide 1 / blood*
  • Humans
  • Insulin / blood
  • Male
  • Middle Aged
  • Pain / etiology
  • Postoperative Complications / blood
  • Postoperative Complications / physiopathology*
  • Postprandial Period
  • Prospective Studies
  • Quality of Life
  • Satiety Response / physiology*
  • Taste Disorders / etiology
  • Treatment Outcome
  • Weight Loss / physiology*

Substances

  • Blood Glucose
  • Insulin
  • Glucagon-Like Peptide 1