Loss of FBXO7 (PARK15) results in reduced proteasome activity and models a parkinsonism-like phenotype in mice

EMBO J. 2016 Sep 15;35(18):2008-25. doi: 10.15252/embj.201593585. Epub 2016 Aug 5.

Abstract

Mutations in the FBXO7 (PARK15) gene have been implicated in a juvenile form of parkinsonism termed parkinsonian pyramidal syndrome (PPS), characterized by Parkinsonian symptoms and pyramidal tract signs. FBXO7 (F-box protein only 7) is a subunit of the SCF (SKP1/cullin-1/F-box protein) E3 ubiquitin ligase complex, but its relevance and function in neurons remain to be elucidated. Here, we report that the E3 ligase FBXO7-SCF binds to and ubiquitinates the proteasomal subunit PSMA2. In addition, we show that FBXO7 is a proteasome-associated protein involved in proteasome assembly. In FBXO7 knockout mice, we find reduced proteasome activity and early-onset motor deficits together with premature death. In addition, we demonstrate that NEX (neuronal helix-loop-helix protein-1)-Cre-induced deletion of the FBXO7 gene in forebrain neurons or the loss of FBXO7 in tyrosine hydroxylase (TH)-positive neurons results in motor defects, reminiscent of the phenotype in PARK15 patients. Taken together, our study establishes a vital role for FBXO7 in neurons, which is required for proper motor control and accentuates the importance of FBXO7 in proteasome function.

Keywords: FBXO7; PARK15; PSMA2; parkinsonism; ubiquitination.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • F-Box Proteins / genetics*
  • F-Box Proteins / metabolism*
  • Gene Knockout Techniques*
  • Mice, Knockout
  • Parkinsonian Disorders / pathology*
  • Proteasome Endopeptidase Complex / metabolism*
  • Protein Processing, Post-Translational
  • Ubiquitination

Substances

  • F-Box Proteins
  • FBXO7 protein, mouse
  • Proteasome Endopeptidase Complex