Prion-Seeding Activity Is widely Distributed in Tissues of Sporadic Creutzfeldt-Jakob Disease Patients

EBioMedicine. 2016 Oct:12:150-155. doi: 10.1016/j.ebiom.2016.08.033. Epub 2016 Aug 24.

Abstract

Human prion diseases are neurodegenerative disorders caused by abnormally folded prion proteins in the central nervous system. These proteins can be detected using the quaking-induced conversion assay. Compared with other bioassays, this assay is extremely sensitive and was used in the present study to determine prion distribution in sporadic Creutzfeldt-Jakob disease patients at autopsy. Although infectivity of the sporadic form is thought to be restricted within the central nervous system, results showed that prion-seeding activities reach 106/g from a 50% seeding dose in non-neuronal tissues, suggesting that prion-seeding activity exists in non-neural organs, and we suggested that non-neural tissues of 106/g SD50 did not exist the infectivity.

Keywords: Creutzfeldt-Jakob disease; Non-neural tissue; Prion; Prion-seeding activity; SD50.

MeSH terms

  • Aged
  • Animals
  • Autopsy
  • Brain / metabolism
  • Case-Control Studies
  • Creutzfeldt-Jakob Syndrome / diagnosis
  • Creutzfeldt-Jakob Syndrome / genetics
  • Creutzfeldt-Jakob Syndrome / metabolism*
  • Creutzfeldt-Jakob Syndrome / pathology*
  • Disease Models, Animal
  • Female
  • Humans
  • Immunohistochemistry
  • Mice
  • Middle Aged
  • Organ Specificity
  • Prion Proteins / genetics
  • Prion Proteins / metabolism*

Substances

  • Prion Proteins

Supplementary concepts

  • Creutzfeldt-Jakob Disease, Sporadic