Targeting flavivirus RNA dependent RNA polymerase through a pyridobenzothiazole inhibitor

Antiviral Res. 2016 Oct:134:226-235. doi: 10.1016/j.antiviral.2016.09.007. Epub 2016 Sep 17.

Abstract

RNA dependent RNA polymerases (RdRp) are essential enzymes for flavivirus replication. Starting from an in silico docking analysis we identified a pyridobenzothiazole compound, HeE1-2Tyr, able to inhibit West Nile and Dengue RdRps activity in vitro, which proved effective against different flaviviruses in cell culture. Crystallographic data show that HeE1-2Tyr binds between the fingers domain and the priming loop of Dengue virus RdRp (Site 1). Conversely, enzyme kinetics, binding studies and mutational analyses suggest that, during the catalytic cycle and assembly of the RdRp-RNA complex, HeE1-2Tyr might be hosted in a distinct binding site (Site 2). RdRp mutational studies, driven by in silico docking analysis, allowed us to locate the inhibition Site 2 in the thumb domain. Taken together, our results provide innovative concepts for optimization of a new class of anti-flavivirus compounds.

Keywords: Crystal structure; Flavivirus; Non-competitive inhibition; Polymerase mutants; RNA dependent RNA polymerase; Virus inhibition.

MeSH terms

  • Antiviral Agents / pharmacology
  • Benzothiazoles / chemistry
  • Benzothiazoles / pharmacology*
  • Binding Sites
  • Catalytic Domain
  • Crystallization
  • Dengue Virus / drug effects
  • Dengue Virus / enzymology
  • Drug Discovery
  • Flavivirus / drug effects
  • Flavivirus / enzymology*
  • Kinetics
  • Models, Molecular
  • Molecular Docking Simulation
  • Mutation
  • RNA-Dependent RNA Polymerase / antagonists & inhibitors*
  • RNA-Dependent RNA Polymerase / drug effects*
  • RNA-Dependent RNA Polymerase / genetics
  • RNA-Dependent RNA Polymerase / metabolism
  • West Nile virus / drug effects
  • West Nile virus / enzymology

Substances

  • Antiviral Agents
  • Benzothiazoles
  • RNA-Dependent RNA Polymerase