IL-15 receptor alpha as the magic wand to boost the success of IL-15 antitumor therapies: The upswing of IL-15 transpresentation

Pharmacol Ther. 2017 Feb:170:73-79. doi: 10.1016/j.pharmthera.2016.10.012. Epub 2016 Oct 21.

Abstract

Interleukin (IL)-15 as a stand-alone therapy can activate the antitumor functions of immune effector cells resulting in significant tumor regression. Interestingly, combining IL-15 with the α-moiety of its receptor (IL-15Rα), also called IL-15 transpresentation, increases the in vivo half-life of IL-15 and enhances binding of IL-15 with cells expressing the IL-15Rβγ, such as NK cells and CD8+ T cells. These features enlarge the signal transmission of IL-15, resulting in improved proliferation and antitumor activities of both NK cells and CD8+ T cells, eventually leading to enhanced killing of tumor cells. In this review, we discuss the antitumor strategies in which this IL-15 transpresentation mechanism is implemented, that are currently under preclinical investigation. Furthermore, we give an overview of the studies in which the IL-15/IL-15Rα complexes are combined with other antitumor therapies. The promising results in these preclinical studies have incited several clinical trials to test the safety and efficacy of IL-15 transpresentation strategies to treat both hematological and advanced solid tumors.

Keywords: Antitumor immunotherapies; Combination therapies; IL-15 receptor alpha; IL-15 transpresentation; Interleukin-15.

Publication types

  • Review
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antineoplastic Agents / administration & dosage
  • Antineoplastic Agents / immunology
  • CD8-Positive T-Lymphocytes / immunology
  • Hematologic Neoplasms / drug therapy
  • Hematologic Neoplasms / immunology
  • Humans
  • Interleukin-15 / administration & dosage*
  • Interleukin-15 / immunology
  • Interleukin-15 Receptor alpha Subunit / immunology*
  • Killer Cells, Natural / immunology
  • Neoplasms / drug therapy*
  • Neoplasms / immunology
  • Neoplasms / pathology

Substances

  • Antineoplastic Agents
  • Interleukin-15
  • Interleukin-15 Receptor alpha Subunit