Netrin-1: A regulator of cancer cell motility?

Eur J Cell Biol. 2016 Nov;95(11):513-520. doi: 10.1016/j.ejcb.2016.10.002. Epub 2016 Oct 19.

Abstract

Netrins form a family of secreted and membrane-associated proteins, netrin-1 being the prototype and most investigated member of the family. The major physiological functions of netrin-1 lie in the regulation of axonal development as well as morphogenesis of different branched organs, by promoting the polarity of migratory/invasive front of the cell. On the other hand, netrin-1 acts as a factor preventing cell apoptosis. These events are mediated via a range of different receptors, including UNC5 and DCC-families. Cancer cells often employ developmental pathways to gain survival and motility advantage. Within recent years, there has been increasing number of observations of upregulation of netrin-1 expression in different forms of cancer, and the increased expression of netrin-1 has been linked to its functions as a survival and invasion promoting factor. We review here recent advances in the netrin-1 related developmental processes that may be of special interest in tumor biology, in addition to the known functions of netrin-1 in tumor biology with special focus on cancer cell migration.

Keywords: Cancer; Cell motility; Cell polarity; Development; Invasion; Netrin.

Publication types

  • Review

MeSH terms

  • Animals
  • Cell Movement*
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Neoplasm Invasiveness
  • Neoplasm Proteins / biosynthesis*
  • Neoplasm Proteins / genetics
  • Neoplasms / genetics
  • Neoplasms / metabolism*
  • Neoplasms / pathology
  • Nerve Growth Factors / biosynthesis*
  • Nerve Growth Factors / genetics
  • Netrin-1
  • Tumor Suppressor Proteins / biosynthesis*
  • Tumor Suppressor Proteins / genetics

Substances

  • NTN1 protein, human
  • Neoplasm Proteins
  • Nerve Growth Factors
  • Tumor Suppressor Proteins
  • Netrin-1