Homozygosity of the Dominant Myotilin c.179C>T (p.Ser60Phe) Mutation Causes a More Severe and Proximal Muscular Dystrophy

J Neuromuscul Dis. 2016 May 27;3(2):275-281. doi: 10.3233/JND-150143.

Abstract

Most myotilinopathy patients present with a dominant late onset distal phenotype and myofibrillar pathology, although the first MYOT mutation in a family reported to have LGMD phenotype. We report here a French family affected with a late onset proximal and distal muscle weakness and myofibrillar myopathy on muscle pathology, in which the siblings known to be clinically affected were homozygous for the c.179C>T (p.Ser60Phe) myotilin gene mutation. One subjectively asymptomatic member of the family was heterozygous for this mutation. This is the first report of a family with patients being homozygous for a known dominant MYOT mutation. Dominant negative mutations are generally considered not to cause a more severe disease in homozygosity, but our data clearly demonstrate the existence of dominant MYOT mutations with a possible dose effect causing a more severe disease phenotype in homozygosity in the spectrum of myofibrillar myopathies (MFM).

Keywords: MYOT; Molecular genetics; c.179C>T (p.Ser60Phe); myofibrillar myopathy; myotilin homozygosity.

MeSH terms

  • Adult
  • Aged
  • Connectin / genetics*
  • Electromyography
  • Female
  • Homozygote*
  • Humans
  • Magnetic Resonance Imaging
  • Male
  • Microfilament Proteins
  • Middle Aged
  • Muscle, Skeletal / diagnostic imaging
  • Muscle, Skeletal / pathology
  • Muscle, Skeletal / physiopathology
  • Muscular Dystrophies / diagnostic imaging
  • Muscular Dystrophies / genetics*
  • Muscular Dystrophies / pathology
  • Muscular Dystrophies / physiopathology
  • Mutation
  • Myopathies, Structural, Congenital / diagnostic imaging
  • Myopathies, Structural, Congenital / genetics*
  • Myopathies, Structural, Congenital / pathology
  • Myopathies, Structural, Congenital / physiopathology
  • Pedigree
  • Severity of Illness Index
  • Siblings

Substances

  • Connectin
  • MYOT protein, human
  • Microfilament Proteins

Supplementary concepts

  • Myofibrillar Myopathy