Abstract
CDK4/6 inhibition was synergistic with dexamethasone and everolimus but antagonistic with conventional chemotherapy in T-cell acute lymphoblastic leukemia (T-ALL) preclinical models. Cyclin-dependent kinase inhibition in combination with glucocorticoids and mTOR inhibition offers a unique therapeutic opportunity in T-ALL. Clin Cancer Res; 23(4); 873-5. ©2016 AACRSee related article by Pikman et al., p. 1012.
©2016 American Association for Cancer Research.
MeSH terms
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Cell Line, Tumor
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Cyclin-Dependent Kinase 4 / antagonists & inhibitors
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Cyclin-Dependent Kinase 4 / genetics
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Dexamethasone / therapeutic use
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Drug Resistance, Neoplasm / drug effects*
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Drug Synergism*
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Everolimus / therapeutic use
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Humans
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Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy*
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Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics
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Precursor Cell Lymphoblastic Leukemia-Lymphoma / pathology
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Precursor T-Cell Lymphoblastic Leukemia-Lymphoma / drug therapy*
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Precursor T-Cell Lymphoblastic Leukemia-Lymphoma / genetics
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Precursor T-Cell Lymphoblastic Leukemia-Lymphoma / pathology
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Protein Kinase Inhibitors / therapeutic use
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Signal Transduction
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TOR Serine-Threonine Kinases / antagonists & inhibitors
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TOR Serine-Threonine Kinases / genetics
Substances
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Protein Kinase Inhibitors
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Dexamethasone
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Everolimus
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MTOR protein, human
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TOR Serine-Threonine Kinases
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CDK4 protein, human
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Cyclin-Dependent Kinase 4