Focusing on frequent ASXL1 mutations in myeloid neoplasms, and considering rarer ASXL2 and ASXL3 mutations
Curr Med Res Opin
.
2017 Apr;33(4):781-782.
doi: 10.1080/03007995.2017.1284049.
Epub 2017 Feb 3.
Authors
Jean S Oak
1
,
Robert S Ohgami
1
Affiliation
1
a Stanford University , Stanford , CA , USA.
PMID:
28097878
DOI:
10.1080/03007995.2017.1284049
No abstract available
Publication types
Editorial
Comment
MeSH terms
Humans
Mutation*
Neoplasms
Repressor Proteins / genetics
Transcription Factors / genetics*
Substances
ASXL1 protein, human
ASXL2 protein, human
ASXL3 protein, human
Repressor Proteins
Transcription Factors