Plaque burden, microstructures and compositions underachieving very low LDL-C levels

Curr Opin Endocrinol Diabetes Obes. 2017 Apr;24(2):122-132. doi: 10.1097/MED.0000000000000317.

Abstract

Purpose of review: To summarize the impact of lowering LDL-C on plaque progression, microstructures and compositions.

Recent findings: Low-density lipoprotein cholesterol (LDL-C) is a major therapeutic target to prevent atherosclerotic cardiovascular disease. Intravascular imaging has elucidated antiatherosclerotic effects of lowering LDL-C in vivo. Intensive control of LDL-C with a statin has been shown to slow plaque progression and induce its regression if very low LDL-C level is achieved. This therapeutic approach has been also demonstrated to modulate plaque microstructures and compositions. These mechanistic insights on intravascular imaging support the benefit of lowering LDL-C in achieving better cardiovascular outcomes.

Summary: Lowering LDL-C level has become the first-line therapy in the primary and secondary prevention settings. The effects of lowering LDL-C on plaque progression, microstructures and compositions will be reviewed in this article.

Publication types

  • Review

MeSH terms

  • Atherosclerosis / drug therapy
  • Atherosclerosis / epidemiology
  • Atherosclerosis / metabolism
  • Atherosclerosis / pathology
  • Cholesterol, LDL / metabolism*
  • Disease Progression
  • Humans
  • Hydroxymethylglutaryl-CoA Reductase Inhibitors / therapeutic use*
  • Plaque, Atherosclerotic / drug therapy*
  • Plaque, Atherosclerotic / epidemiology
  • Plaque, Atherosclerotic / pathology*
  • Plaque, Atherosclerotic / ultrastructure*

Substances

  • Cholesterol, LDL
  • Hydroxymethylglutaryl-CoA Reductase Inhibitors