Bombesin Antagonist-Based Radiotherapy of Prostate Cancer Combined with WST-11 Vascular Targeted Photodynamic Therapy

Clin Cancer Res. 2017 Jul 1;23(13):3343-3351. doi: 10.1158/1078-0432.CCR-16-2745. Epub 2017 Jan 20.

Abstract

Purpose: DOTA-AR, a bombesin-antagonist peptide, has potential clinical application for targeted imaging and therapy in gastrin-releasing peptide receptor (GRPr)-positive malignancies when conjugated with a radioisotope such as 90Y. This therapeutic potential is limited by the fast washout of the conjugates from the target tumors. WST-11 (Weizmann STeba-11 drug; a negatively charged water-soluble palladium-bacteriochlorophyll derivative, Tookad Soluble) vascular targeted photodynamic therapy (VTP) is a local ablation approach recently approved for use in early-stage prostate cancer. It generates reactive oxygen/nitrogen species within tumor blood vessels, resulting in their instantaneous destruction followed by rapid tumor necrosis. We hypothesize that the instantaneous arrest of tumor vasculature may provide a means to trap radiopharmaceuticals within the tumor, thereby improving the efficacy of targeted radiotherapy.Experimental Design: GRPr-positive prostate cancer xenografts (PC-3 and VCaP) were treated with 90Y-DOTA-AR with or without VTP. The uptake of radioisotopes was monitored by Cherenkov luminescence imaging (CLI). The therapeutic efficacy of the combined VTP and 90Y-DOTA-AR in PC-3 xenografts was assessed.Results: CLI of 90Y-DOTA-AR demonstrated longer retention of radiotracer within the VTP-treated PC-3 xenografts compared with the non-VTP-treated ones (P < 0.05) at all time points (24-144 hours) after 90Y-DOTA-AR injection. A similar pattern of retention was observed in VCaP xenografts. When 90Y-DOTA-AR administration was combined with VTP, tumor growth delay was significantly longer than for the control or the monotherapy groups.Conclusions: Tumor vascular arrest by VTP improves 90Y-DOTA-AR retention in the tumor microenvironment thereby enhancing therapeutic efficacy. Clin Cancer Res; 23(13); 3343-51. ©2017 AACR.

MeSH terms

  • Animals
  • Bacteriochlorophylls / administration & dosage
  • Bombesin / administration & dosage*
  • Bombesin / antagonists & inhibitors
  • Cell Line, Tumor
  • Cell Proliferation / drug effects*
  • Cell Proliferation / radiation effects
  • Humans
  • Male
  • Mice
  • Peptides / administration & dosage*
  • Photochemotherapy
  • Prostatic Neoplasms / drug therapy*
  • Prostatic Neoplasms / pathology
  • Prostatic Neoplasms / radiotherapy
  • Radioisotopes / administration & dosage
  • Tissue Distribution / drug effects
  • Tissue Distribution / radiation effects
  • Tumor Microenvironment / drug effects
  • Tumor Microenvironment / radiation effects
  • Xenograft Model Antitumor Assays

Substances

  • Bacteriochlorophylls
  • Peptides
  • Radioisotopes
  • padeliporfin
  • Bombesin