Effects of ginsenosides, lectins and Momordica charantia insulin-like peptide on corticosterone production by isolated rat adrenal cells

J Ethnopharmacol. 1987 Sep-Oct;21(1):21-9.

Abstract

Ginsenosides Rb1, Rb2, Rc and Rg1 inhibited steroidogenesis induced by a maximally active dose of corticotropin in isolated rat adrenal cells. The galactose-binding lectins from Momordica charantia seeds and Trichosanthes kirilowii tubers and mannose-binding concanavalin A did not affect basal corticosterone production. The lectins potentiated steroidogenesis induced by a submaximal dose of corticotropin but were without effect on steroidogenesis induced by a maximally active dose of corticotropin. Momordica charantia insulin-like peptide did not affect steroidogenesis.

MeSH terms

  • Adrenal Glands / drug effects
  • Adrenal Glands / metabolism*
  • Adrenocorticotropic Hormone / metabolism
  • Animals
  • Cells, Cultured
  • Concanavalin A / pharmacology
  • Corticosterone / biosynthesis*
  • Ginsenosides
  • Insulin / pharmacology
  • Lectins / pharmacology*
  • Male
  • Peptides / pharmacology*
  • Plant Lectins
  • Plants, Medicinal / analysis*
  • Radioimmunoassay
  • Rats
  • Rats, Inbred Strains
  • Saponins / pharmacology*

Substances

  • Ginsenosides
  • Insulin
  • Lectins
  • Peptides
  • Plant Lectins
  • Saponins
  • Concanavalin A
  • Adrenocorticotropic Hormone
  • Corticosterone