Damaged rat peripheral nerves do not contain detectable amounts of alpha-MSH

J Neurosci Res. 1988;19(1):14-8. doi: 10.1002/jnr.490190103.

Abstract

Peptides related to alpha-MSH (collectively termed melanocortins) stimulate nerve growth following injury and may play a physiological role in the repair process. Melanocortins are not normally present in mature peripheral nerves but MSH-like bioactivity has been observed in extracts of injured nerves. alpha-MSH could derive from reexpression of the POMC prohormone in injured nerves or from proteolysis of the intermediate-size neurofilament protein that bears antigenic similarities to melanocortins. Using a radioimmunoassay that will distinguish between alpha-MSH and neurofilament-derived fragments, we have shown that alpha-MSH is not present (detection limit 74 pg alpha-MSH/mg protein) in damaged rat sciatic nerves.

MeSH terms

  • Animals
  • Female
  • Immunohistochemistry
  • Nerve Crush
  • Nerve Degeneration*
  • Radioimmunoassay
  • Rats
  • Rats, Inbred Strains
  • Sciatic Nerve / metabolism
  • Sciatic Nerve / physiology*
  • alpha-MSH / metabolism*

Substances

  • alpha-MSH