Development of a 18F-Labeled Radiotracer with Improved Brain Kinetics for Positron Emission Tomography Imaging of Translocator Protein (18 kDa) in Ischemic Brain and Glioma

J Med Chem. 2017 May 11;60(9):4047-4061. doi: 10.1021/acs.jmedchem.7b00374. Epub 2017 May 1.

Abstract

We designed four novel acetamidobenzoxazolone compounds 7a-d as candidates for positron emission tomography (PET) radiotracers for imaging the translocator protein (18 kDa, TSPO) in ischemic brain and glioma. Among these compounds, 2-(5-(6-fluoropyridin-3-yl)-2-oxobenzo[d]oxazol-3(2H)-yl)-N-methyl-N-phenylacetamide (7d) exhibited high binding affinity (Ki = 13.4 nM) with the TSPO and moderate lipophilicity (log D = 1.92). [18F]7d was radiosynthesized by [18F]fluorination of the bromopyridine precursor 7h with [18F]F- in 12 ± 5% radiochemical yield (n = 6, decay-corrected). In vitro autoradiography and PET studies of ischemic rat brain revealed higher binding of [18F]7d with TSPO on the ipsilateral side, as compared to the contralateral side, and improved brain kinetics compared with our previously developed radiotracers. Metabolite study of [18F]7d showed 93% of unchanged form in the ischemic brain at 30 min after injection. Moreover, PET study with [18F]7d provided a clear tumor image in a glioma-bearing rat model. We demonstrated that [18F]7d is a useful PET radiotracer for visualizing not only neuroinflammation but also glioma and will translate this radiotracer to a "first-in-human" study in our facility.

MeSH terms

  • Animals
  • Brain / metabolism*
  • Brain Ischemia / metabolism*
  • Brain Neoplasms / metabolism*
  • Carrier Proteins / metabolism*
  • Fluorine Radioisotopes / pharmacokinetics*
  • Glioma / metabolism*
  • Positron-Emission Tomography*
  • Rats
  • Receptors, GABA-A / metabolism*

Substances

  • Carrier Proteins
  • Fluorine Radioisotopes
  • Receptors, GABA-A
  • Tspo protein, rat