Dying to protect: cell death and the control of T-cell homeostasis

Immunol Rev. 2017 May;277(1):21-43. doi: 10.1111/imr.12538.

Abstract

T cells play a critical role in immune responses as they specifically recognize peptide/MHC complexes with their T-cell receptors and initiate adaptive immune responses. While T cells are critical for performing appropriate effector functions and maintaining immune memory, they also can cause autoimmunity or neoplasia if misdirected or dysregulated. Thus, T cells must be tightly regulated from their development onward. Maintenance of appropriate T-cell homeostasis is essential to promote protective immunity and limit autoimmunity and neoplasia. This review will focus on the role of cell death in maintenance of T-cell homeostasis and outline novel therapeutic strategies tailored to manipulate cell death to limit T-cell survival (eg, autoimmunity and transplantation) or enhance T-cell survival (eg, vaccination and immune deficiency).

Keywords: T cells; apoptosis/autophagy; autoimmunity; infectious diseases; memory.

Publication types

  • Review
  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Autoimmune Diseases / immunology*
  • Autoimmune Diseases / therapy
  • Cell Death
  • Cell Survival
  • Graft Rejection / immunology*
  • Graft Rejection / prevention & control
  • Homeostasis
  • Humans
  • Immunologic Deficiency Syndromes / immunology*
  • Immunologic Deficiency Syndromes / therapy
  • Immunotherapy / methods*
  • T-Lymphocytes / physiology*
  • Transplantation
  • Vaccination