Neurons and neuronal activity control gene expression in astrocytes to regulate their development and metabolism

Nat Commun. 2017 May 2:8:15132. doi: 10.1038/ncomms15132.

Abstract

The influence that neurons exert on astrocytic function is poorly understood. To investigate this, we first developed a system combining cortical neurons and astrocytes from closely related species, followed by RNA-seq and in silico species separation. This approach uncovers a wide programme of neuron-induced astrocytic gene expression, involving Notch signalling, which drives and maintains astrocytic maturity and neurotransmitter uptake function, is conserved in human development, and is disrupted by neurodegeneration. Separately, hundreds of astrocytic genes are acutely regulated by synaptic activity via mechanisms involving cAMP/PKA-dependent CREB activation. This includes the coordinated activity-dependent upregulation of major astrocytic components of the astrocyte-neuron lactate shuttle, leading to a CREB-dependent increase in astrocytic glucose metabolism and elevated lactate export. Moreover, the groups of astrocytic genes induced by neurons or neuronal activity both show age-dependent decline in humans. Thus, neurons and neuronal activity regulate the astrocytic transcriptome with the potential to shape astrocyte-neuron metabolic cooperation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Astrocytes / cytology
  • Astrocytes / metabolism*
  • CREB-Binding Protein / genetics
  • CREB-Binding Protein / metabolism
  • Cell Communication
  • Cerebral Cortex / cytology
  • Cerebral Cortex / growth & development
  • Cerebral Cortex / metabolism*
  • Coculture Techniques
  • Cyclic AMP / metabolism
  • Cyclic AMP-Dependent Protein Kinases / genetics
  • Cyclic AMP-Dependent Protein Kinases / metabolism
  • Disease Models, Animal
  • Embryo, Mammalian
  • Gene Expression Profiling
  • Gene Expression Regulation, Developmental*
  • Glucose / metabolism
  • High-Throughput Nucleotide Sequencing
  • Humans
  • Lactic Acid / metabolism
  • Membrane Potentials / physiology
  • Mice, Knockout
  • Neurons / cytology
  • Neurons / metabolism*
  • Rats, Sprague-Dawley
  • Receptors, Notch / genetics
  • Receptors, Notch / metabolism
  • Signal Transduction
  • Tauopathies / genetics*
  • Tauopathies / metabolism
  • Tauopathies / pathology

Substances

  • Receptors, Notch
  • Lactic Acid
  • Cyclic AMP
  • CREB-Binding Protein
  • Crebbp protein, mouse
  • Cyclic AMP-Dependent Protein Kinases
  • Glucose