Th17/Treg dysregulation in allergic asthmatic children is associated with elevated notch expression

J Asthma. 2018 Jan;55(1):1-7. doi: 10.1080/02770903.2016.1266494. Epub 2017 May 16.

Abstract

Background: Notch signaling pathway is critically involved in the differentiation of T helper (Th) cells, key players in the pathogenesis of allergic diseases.

Objective: The study is to explore whether Th17/Treg dysregulation in children with allergic asthma (AA) is associated with alteration of Notch expression.

Methods: Thirty-five patients with AA and thirty-five healthy control children were selected. Flow cytometry was used to detect Th17 and Treg cells. Quantitative real-time polymerase chain reaction (QRT-PCR) was used to measure the expression of Notch1 mRNA. The correlations among Notch1 mRNA expression, the percentage of Th17 cells, and Th17/Treg ratio were calculated.

Results: Th17 and Treg cells were significantly increased and decreased, respectively, in children with AA than in healthy control (p < 0.001). mRNA level of Notch1 was elevated in children with AA comparing to healthy controls (p < 0.001). The mRNA expression of Notch1 was positively correlated with the percentage of Th17 cells (r = 0.775, p < 0.001) and Th17/Treg ratio (r = 0.698, p < 0.001).

Conclusion: Children with AA showed dysregulation of Th17/Treg cells in peripheral blood. Such change is accompanied with overexpression of Notch1, indicating Th17/Treg dysregulation in children with AA is associated with elevated Notch expression.

Keywords: Asthma; Notch signaling pathway; Th17; Treg; children.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Asthma / blood
  • Asthma / immunology*
  • Case-Control Studies
  • Cell Differentiation / immunology
  • Child
  • Child, Preschool
  • Female
  • Flow Cytometry
  • Humans
  • Interleukin-17 / immunology
  • Interleukin-17 / metabolism
  • Male
  • RNA, Messenger / metabolism
  • Receptor, Notch1 / genetics
  • Receptor, Notch1 / metabolism*
  • Signal Transduction / immunology*
  • T-Lymphocytes, Regulatory / immunology*
  • T-Lymphocytes, Regulatory / metabolism
  • Th17 Cells / immunology*
  • Th17 Cells / metabolism

Substances

  • Interleukin-17
  • NOTCH1 protein, human
  • RNA, Messenger
  • Receptor, Notch1