Short Overview

Methods Mol Biol. 2018:1759:3-8. doi: 10.1007/7651_2017_38.

Abstract

Mitochondrial autophagy (mitophagy) is a mitochondrial quality control mechanism that selectively removes damaged mitochondria via autophagic degradation. Autophagic adaptor/receptor proteins contribute to the selective degradation of damaged mitochondria by autophagy. A part of them containing both ubiquitin binding domains and Atg8 interacting motif (AIM)/LC3 interacting region (LIR) motifs, which bind to the autophagy-related protein 8 (Atg8) family (LC3 and GABARAP family), lead ubiquitylated (damaged) mitochondria to selective removal. On the other hand, some specific outer mitochondrial membrane-anchored proteins containing AIM/LIR motif function as another type of autophagy adaptor/receptor proteins. Here I briefly summarize mechanisms of mitophagy and its related proteins.

Keywords: Atg8 interacting motif (AIM)/LC3 interacting region (LIR); Autophagy adaptor; Mitophagy.

Publication types

  • Review

MeSH terms

  • Animals
  • Autophagy
  • Biomarkers
  • Humans
  • Mitochondria / genetics
  • Mitochondria / metabolism
  • Mitophagy*
  • Yeasts / genetics
  • Yeasts / metabolism

Substances

  • Biomarkers