Abstract
The loss of cone photoreceptors that mediate daylight vision represents a leading cause of blindness, for which cell replacement by transplantation offers a promising treatment strategy. Here, we characterize cone differentiation in retinas derived from mouse embryonic stem cells (mESCs). Similar to in vivo development, a temporal pattern of progenitor marker expression is followed by the differentiation of early thyroid hormone receptor β2-positive precursors and, subsequently, photoreceptors exhibiting cone-specific phototransduction-related proteins. We establish that stage-specific inhibition of the Notch pathway increases cone cell differentiation, while retinoic acid signaling regulates cone maturation, comparable with their actions in vivo. MESC-derived cones can be isolated in large numbers and transplanted into adult mouse eyes, showing capacity to survive and mature in the subretinal space of Aipl1-/- mice, a model of end-stage retinal degeneration. Together, this work identifies a robust, renewable cell source for cone replacement by purified cell suspension transplantation.
Keywords:
blindness; cell- and tissue-based therapy; mouse embryonic stem cells; retina; retinal cone photoreceptor cells; retinal degeneration.
Copyright © 2017 The Authors. Published by Elsevier Inc. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Adaptor Proteins, Signal Transducing / deficiency
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Adaptor Proteins, Signal Transducing / genetics
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Animals
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Basic-Leucine Zipper Transcription Factors / antagonists & inhibitors
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Basic-Leucine Zipper Transcription Factors / genetics
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Basic-Leucine Zipper Transcription Factors / metabolism
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Cell Differentiation / drug effects
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Disease Models, Animal
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Eye Proteins / antagonists & inhibitors
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Eye Proteins / genetics
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Eye Proteins / metabolism
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Hepatocyte Nuclear Factor 6 / metabolism
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Leukemia Inhibitory Factor / pharmacology
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Mice
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Mice, Knockout
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Mouse Embryonic Stem Cells / cytology
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Mouse Embryonic Stem Cells / transplantation*
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Oligodendrocyte Transcription Factor 2 / metabolism
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Opsins / metabolism
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Orphan Nuclear Receptors / antagonists & inhibitors
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Orphan Nuclear Receptors / genetics
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Orphan Nuclear Receptors / metabolism
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Otx Transcription Factors / metabolism
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RNA Interference
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RNA, Small Interfering / metabolism
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Receptors, Notch / antagonists & inhibitors
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Receptors, Notch / metabolism
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Retinal Cone Photoreceptor Cells / cytology*
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Retinal Cone Photoreceptor Cells / metabolism
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Retinal Degeneration / pathology
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Retinal Degeneration / therapy*
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Signal Transduction
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Tretinoin / metabolism
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Tretinoin / pharmacology
Substances
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Adaptor Proteins, Signal Transducing
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Aipl1 protein, mouse
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Basic-Leucine Zipper Transcription Factors
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Eye Proteins
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Hepatocyte Nuclear Factor 6
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Leukemia Inhibitory Factor
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Nr2e3 protein, mouse
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Nrl protein, mouse
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Olig2 protein, mouse
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Oligodendrocyte Transcription Factor 2
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Onecut1 protein, mouse
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Opsins
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Orphan Nuclear Receptors
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Otx Transcription Factors
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Otx2 protein, mouse
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RNA, Small Interfering
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Receptors, Notch
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Tretinoin