GRP78 Participates in PCA3-regulated Prostate Cancer Progression

Anticancer Res. 2017 Aug;37(8):4303-4310. doi: 10.21873/anticanres.11823.

Abstract

Background/aim: The human prostate cancer antigen 3 (PCA3) is a long non-coding RNA (lncRNA) commonly used as a diagnostic marker for prostate cancer (PCa). Herein we investigated the cellular function of PCA3 in PCa and its potential mechanism.

Materials and methods: PCA3 was overexpressed in a PC3 cell line (PC3PCA+) and cell proliferation, migration, invasion and apoptosis were compared to those of control cells (PC3NC). Differentially expressed proteins were identified by two-dimensional polyacrylamide gel electrophoresis (2D-PAGE) and mass spectrometry.

Results: Overexpression of PCA3 significantly increased cell proliferation rate, migration and invasion, while inhibited apoptosis in PC3 cells. Three proteins were found down-regulated and 7 proteins up-regulated in PC3PCA+ cells compared to PC3NC cells, including GRP78. Higher GRP78 was also found in PCa clinical specimens.

Conclusion: This study confirmed that in PCa, PCA3 plays a pro-cancer role through promoting cell proliferation, migration and invasion while inhibiting cell apoptosis. This process might involve the up-regulation of GRP78.

Keywords: GRP78; PCA3; Prostate cancer; cancer-related genes; long non-coding RNA.

MeSH terms

  • Antigens, Neoplasm / genetics*
  • Cell Line, Tumor
  • Cell Movement
  • Cell Proliferation
  • Disease Progression
  • Endoplasmic Reticulum Chaperone BiP
  • Gene Expression Regulation, Neoplastic
  • Heat-Shock Proteins / genetics*
  • Heat-Shock Proteins / metabolism*
  • Humans
  • Male
  • Neoplasm Invasiveness
  • Prostatic Neoplasms / genetics*
  • Prostatic Neoplasms / metabolism

Substances

  • Antigens, Neoplasm
  • Endoplasmic Reticulum Chaperone BiP
  • HSPA5 protein, human
  • Heat-Shock Proteins
  • prostate cancer antigen 3, human