[Effect of Heat Shock Protein 90 Inhibitor 17-DMAG on Proliferation and Apoptosis of Acute Lymphocytic Leukemia Cell Line Jurkat]

Zhongguo Shi Yan Xue Ye Xue Za Zhi. 2017 Aug;25(4):1011-1015. doi: 10.7534/j.issn.1009-2137.2017.04.009.
[Article in Chinese]

Abstract

Objective: To explore the effect of heat shock protein 90(HSP90) inhibitor 17-DMAG, an inhibitor specific for heat shock protein 90, on the proliferation and apoptosis of acute lymphocytic leukemia cell lines Jurkat.

Methods: Jurkat cells were collected, then were treated with 17-DMAG. The expression of HSP90 was examined by semi-quantitative RT-PCR analysis, the effect of 17-DMAG on cell proliferation were detected by using WST, and cell apoptosis were detected by using flow cytometry with Annexin V/PI double stenining.

Results: After Jurkat cells were treated with different concentrations of 17-DMAG for 48 hours, the HSP90 mRNA expression decreased significantly in dose dependent manner (r=0.9530, P<0.01). The IC50 was 3.17 mmol/L when the Jurkat cells were treated with 17-DMAG for 48 h; after treating Jurkat cell with 17-DMAG, the cell proliferation was inhibited(r=0.9903, P< 0.01), the cell apoptosis was increased in dose dependent manner (r=0.9876, P<0.01).

Conclusion: 17-DMAG can inhibit the Jurkat cell proliferation and induce the Jurkat cell apoptosis.

MeSH terms

  • Apoptosis
  • Benzoquinones
  • Cell Line
  • Cell Proliferation
  • HSP90 Heat-Shock Proteins
  • Humans
  • Jurkat Cells
  • Lactams, Macrocyclic
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma*

Substances

  • Benzoquinones
  • HSP90 Heat-Shock Proteins
  • Lactams, Macrocyclic
  • 17-(dimethylaminoethylamino)-17-demethoxygeldanamycin