The Prohormone VGF Regulates β Cell Function via Insulin Secretory Granule Biogenesis

Cell Rep. 2017 Sep 5;20(10):2480-2489. doi: 10.1016/j.celrep.2017.08.050.

Abstract

The prohormone VGF is expressed in neuroendocrine and endocrine tissues and regulates nutrient and energy status both centrally and peripherally. We and others have shown that VGF-derived peptides have direct action on the islet β cell as secretagogues and cytoprotective agents; however, the endogenous function of VGF in the β cell has not been described. Here, we demonstrate that VGF regulates secretory granule formation. VGF loss-of-function studies in both isolated islets and conditional knockout mice reveal a profound decrease in stimulus-coupled insulin secretion. Moreover, VGF is necessary to facilitate efficient exit of granule cargo from the trans-Golgi network and proinsulin processing. It also functions to replenish insulin granule stores following nutrient stimulation. Our data support a model in which VGF operates at a critical node of granule biogenesis in the islet β cell to coordinate insulin biosynthesis with β cell secretory capacity.

Keywords: VGF; chromogranin B; granule biogenesis; insulin secretion; secretory granule; β cell function.

MeSH terms

  • Animals
  • Cytoplasmic Granules / metabolism
  • Glucose / metabolism
  • Insulin / metabolism*
  • Insulin-Secreting Cells / metabolism*
  • Islets of Langerhans / metabolism
  • Mice
  • Mice, Knockout
  • Neuropeptides / genetics
  • Neuropeptides / metabolism
  • Secretory Vesicles / metabolism*
  • trans-Golgi Network / metabolism

Substances

  • Insulin
  • Neuropeptides
  • VGF peptide
  • Glucose