Elongation Factor P and the Control of Translation Elongation

Annu Rev Microbiol. 2017 Sep 8:71:117-131. doi: 10.1146/annurev-micro-090816-093629. Epub 2017 May 22.

Abstract

Elongation factor P (EF-P) binds to ribosomes requiring assistance with the formation of oligo-prolines. In order for EF-P to associate with paused ribosomes, certain tRNAs with specific d-arm residues must be present in the peptidyl site, e.g., tRNAPro. Once EF-P is accommodated into the ribosome and bound to Pro-tRNAPro, productive synthesis of the peptide bond occurs. The underlying mechanism by which EF-P facilitates this reaction seems to have entropic origins. Maximal activity of EF-P requires a posttranslational modification in Escherichia coli, Pseudomonas aeruginosa, and Bacillus subtilis. Each of these modifications is distinct and ligated onto its respective EF-P through entirely convergent means. Here we review the facets of translation elongation that are controlled by EF-P, with a particular focus on the purpose behind the many different modifications of EF-P.

Keywords: posttranslational modification; proline; protein synthesis; ribosome.

Publication types

  • Review

MeSH terms

  • Bacillus subtilis / enzymology*
  • Bacillus subtilis / genetics
  • Escherichia coli / enzymology*
  • Escherichia coli / genetics
  • Peptide Chain Elongation, Translational*
  • Peptide Elongation Factors / metabolism*
  • Protein Binding
  • Pseudomonas aeruginosa / enzymology*
  • Pseudomonas aeruginosa / genetics
  • RNA, Transfer, Pro / metabolism
  • Ribosomes / metabolism

Substances

  • Peptide Elongation Factors
  • RNA, Transfer, Pro
  • factor EF-P