A systematic integrated analysis of brain expression profiles reveals YAP1 and other prioritized hub genes as important upstream regulators in Alzheimer's disease

Alzheimers Dement. 2018 Feb;14(2):215-229. doi: 10.1016/j.jalz.2017.08.012. Epub 2017 Sep 18.

Abstract

Introduction: Profiling the spatial-temporal expression pattern and characterizing the regulatory networks of brain tissues are vital for understanding the pathophysiology of Alzheimer's disease (AD).

Methods: We performed a systematic integrated analysis of expression profiles of AD-affected brain tissues (684 AD and 562 controls). A network-based convergent functional genomic approach was used to prioritize possible regulator genes during AD development, followed by functional characterization.

Results: We generated a complete list of differentially expressed genes and hub genes of the transcriptomic network in AD brain and constructed a Web server (www.alzdata.org) for public access. Seventeen hub genes active at the early stages, especially YAP1, were recognized as upstream regulators of the AD network. Cellular assays proved that early alteration of YAP1 could promote AD by influencing the whole transcriptional network.

Discussion: Early expression disturbance of hub genes is an important feature of AD development, and interfering with this process may reverse the disease progression.

Keywords: Alzheimer's disease; Convergent functional genomic; Database; Differential expression; Upstream regulator.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptor Proteins, Signal Transducing / genetics*
  • Adaptor Proteins, Signal Transducing / metabolism
  • Alzheimer Disease / diagnostic imaging
  • Alzheimer Disease / genetics*
  • Alzheimer Disease / pathology
  • Alzheimer Disease / physiopathology*
  • Brain / diagnostic imaging
  • Brain / metabolism*
  • Databases, Genetic / statistics & numerical data
  • Disease Progression
  • Female
  • Gene Expression / physiology*
  • Gene Regulatory Networks / physiology*
  • Genomics
  • Humans
  • Male
  • Phosphoproteins / genetics*
  • Phosphoproteins / metabolism
  • Transcription Factors
  • YAP-Signaling Proteins

Substances

  • Adaptor Proteins, Signal Transducing
  • Phosphoproteins
  • Transcription Factors
  • YAP-Signaling Proteins
  • YAP1 protein, human