Identification and characterization of interferon signaling-related microRNAs in occult hepatitis B virus infection

Clin Epigenetics. 2017 Sep 16:9:101. doi: 10.1186/s13148-017-0404-9. eCollection 2017.

Abstract

Background: Occult hepatitis B virus infection (OBI) is an important risk factor of liver cirrhosis and hepatocellular carcinoma. Type 1 interferon (IFN) signaling-related miRNAs were significantly associated with hepatitis B virus (HBV) infection. However, the characteristics of serum IFN signaling-related miRNAs in OBI remain unclear. Therefore, this study aimed to analyze the expression levels of serum IFN signaling-related miRNAs in OBI and to evaluate their potential values for OBI diagnosis.

Methods: Twenty serum samples for training test (10 healthy controls and 10 OBI patients) and 438 validation serum samples from healthy controls, asymptomatic HBsAg carriers (ASC), and chronic hepatitis B (CHB) and OBI patients were collected. Expression levels of 32 IFN signaling-related miRNAs were analyzed in training and validation sets of samples using RT-qPCR.

Results: Among 32 IFN signaling-related miRNAs, decreased miR-122 levels and increased miR-130a levels were detected in training OBI samples. Furthermore, the results from validation test showed that the mean serum miR-122 and miR-130a level was 2.28 ± 0.96 and 3.11 ± 0.93 in OBI subjects, respectively. Compared to the healthy controls, ASC and CHB patients, miR-122 levels were significantly downregulated, while miR-130a levels were significantly upregulated in OBI patients. ROC analysis indicated that miR-122 + miR-130a could differentiate OBI from healthy controls, ASC, and CHB (≥ 0.87 of AUC).

Conclusions: Our study suggested that decreased serum miR-122 level and increased miR-130a level were significantly associated with OBI. Moreover, a combination of miR-122 and miR-130a could be served as a potential marker for OBI diagnosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Case-Control Studies
  • DNA Methylation
  • Female
  • Gene Expression Regulation*
  • Genetic Predisposition to Disease
  • Hepatitis B / blood
  • Hepatitis B / diagnosis
  • Hepatitis B / genetics*
  • Humans
  • Interferon Type I / genetics
  • Male
  • MicroRNAs / blood*
  • Middle Aged
  • Signal Transduction

Substances

  • Interferon Type I
  • MIRN122 microRNA, human
  • MIRN130 microRNA, human
  • MicroRNAs